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cells
Xi defined a partition ofWf into canonical right cells and the right order ≤R on the set of cells.
      
This paper shows how the Kazhdan-Lusztig theory of cells can be directly applied to establish the quasi-heredity ofq-Schur algebras.
      
Coordinates on Schubert cells, Kostant's harmonic forms, and the Bruhat Poisson structure onG/B
      
Also, we discuss possible connections to the positive and cluster geometry of G/B+ × G/B-, which would generalize results of Fomin and Zelevinsky on double Bruhat cells and Marsh and Rietsch on double Schubert cells.
      
Alpha2-Adrenergic Receptors in Intestinal Epithelial Cells: Mechanisms of Signaling, Role, and Regulation
      
Alpha2-Adrenergic Receptors in Intestinal Epithelial Cells: Mechanisms of Signaling, Role, and Regulation
      
In the present study, two of the probable an umor marine compounds, manzamine A and sarcophine, were screened using benzo[a]pyrene (BP)-derived DNA adduct formation in MCF-7 cells as intermediary biomarker.
      
Briefly, MCF-7 cells were treated with the compounds for 24 h followed by treatment with BP (0.5 μM).
      
The Histamine H4 receptor is expressed mainly on eosinophils and mast cells and has been shown to be involved in the chemotaxis of both the cell types.
      
The receptor is also implicated in the release of IL-16, from CD8+ T cells.
      
Their cytotoxic properties were determined by a 3-day continuous exposure MTT assay with murine melanoma B16 cells.
      
fenestratum were evaluated for cytotoxicity on L929 fibroblast cells.
      
Methanol extracts demonstrated better inhibitory effects on cultured L929 cells followed by purified berberine from cell suspension cultures and water extracts.
      
Only 15.33% of the cells was metabolically active at 200 μg/ml methanol extract while 23.57% of the cells was active at 200 μg/ml berberine.
      
Most of the compounds increase the cAMP content of CHO cells expressing the human A1-adenosine receptor, indicating an antagonist activity.
      
Seven compounds caused 50% growth inhibition (GI50) of tumor cells at concentrations of >amp;lt;100 μM while the remaining ten were not cytotoxic.
      
Inhibitive Effect of Prodigiosin on the Proliferation of Human Malignant Pancreatic Cancer Cells
      
In this study, we find that prodigiosin could effectively inhibit the proliferation of human pancreatic cancer cells H8898 in a dose-and-time-dependent manner, with an IC50 of 75μmol according to the results of MTT and cell proliferation assays.
      
Prodigiosin also could induce apoptosis of pancreatic cancer cells at low concentration and results in the fragmentation pattern of DNA.
      
Prodigiosin may effectively enter cells and promote the level of intracellular reactive oxygen species (ROSin) in a dose-dependent manner.
      
 

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