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Acute motor axonal neuropathy presenting with bowel, bladder, and erectile dysfunction
      
Day wetting children had a significantly higher rate of previous antibiotic prophylaxis, larger residual volume, thicker bladder walls; the uroflow curves were significantly less bell- and more staccato-shaped, the EMG less relaxed.
      
Aminergic innervation of the gall bladder in man and dog
      
Human gall bladder also contains a population of putatively tryptaminergic, intramural neuron-like cells.
      
However, there was no evidence for tryptaminergic innervation of effector structures in the gall bladder wall.
      
In the female subjects, the probe was advanced into the bladder and the procedure repeated, and the gasp resulted in 60% and 83% drop in flux.
      
Measurements were made of voiding time, time for 50% emptying, average flow rate, ejection fraction, post-void residual urine volume, voided volume, and total bladder volume.
      
Six tests demonstrated pupillary, lacrimal, salivary, urinary bladder, sexual and sudomotor dysfunction in the majority of patients.
      
Subcutaneous administration of the parasympathomimetic agent bethanechol demonstrated hyperresponsiveness of lacrimal, salivary, oesophageal, bowel, bladder and sudomotor functions.
      
Modest increases in urinary bladder pressure result in acute hypertensive episodes in humans with spinal cord lesions above T5.
      
The aim of this study was to characterize the contribution of alpha- and beta-adrenoceptors as well as circulating neuropeptide-Y (NPY) to the pressor response to bladder distension in conscious cervical spinal rats.
      
Blood samples were taken before, during and after bladder distension for determination of plasma NPY by radioimmunoassay.
      
The pressor response to bladder distension was approximately 30 mmHg under control conditions.
      
There were no changes in plasma NPY in response to bladder distension.
      
Moreover, both α and β adrenergic receptors contribute to the pressor response induced by bladder distension in the conscious cervical spinal rat.
      
Conventional head-up tilt testing with an empty urinary bladder caused no change in arterial blood pressure, but a moderate increase in heart rate.
      
Urinary bladder filling caused minimal increases of the arterial pressure and heart rate.
      
The sitting posture with a distended bladder caused mild orthostatic hypotension.
      
Urinary bladder evacuation caused a fall in arterial pressure with a decrease in heart rate.
      
Simultaneous arterial and urinary bladder pressure recordings in multiple system atrophy and in spinal disorders with detrusor h
      
 

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