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biopsy specimens
The immunohistological analysis of the endomyocardial biopsy specimens revealed pathologically increased lymphocytic infiltrates and increased expression of interstitial and endothelial MHC I and II antigens.
      
Biopsy specimens were examined with the light and electron microscope.
      
Fibrillar astrocytomas in man, as studied in biopsy specimens from the cerebral hemispheres in two cases, reveal uniform distribution and isomorphic fine structure of the tumor cells.
      
44 biopsy specimens from cases with muscular dystrophy or myositis were evaluated applying schematic criteria to facilitate differential diagnosis.
      
Biopsy specimens of 15 cases of Morton's metatarsalgia of varible durations of illness were examined neurohistologically.
      
Clinical peculiarities as well as alterations of biopsy specimens taken from skin and muscle are pointed out.
      
This increased lysosomal reaction was demonstrated also in biochemical analyses performed on MS biopsy specimens.
      
Myopathic patterns with various histological changes in muscle biopsy specimens were seen in all eight reported cases of the rigid spine syndrome, but there are no characteristic histological findings in the rigid spine syndrome.
      
Congo red staining and polarization microscopy of biopsy specimens revealed amyloid deposits.
      
Two patients had a slightly reduced nerve conduction velocity and in two cases moderate abnormalities were found in muscle biopsy specimens.
      
Liver-biopsy specimens from 10 MND patients were compared by electron microscopic examination with specimens from age-matched controls who had chronic persistent hepatitis.
      
In five skin biopsy specimens of different regions typical vacuoles were noted in Schwann cells, histiocytes, nevus cells, and rarely in perineurial cells.
      
In biopsy specimens from the patients with laryngeal carcinoma, neurogenic atrophy and various structural changes in the muscle fibres were the prominent features.
      
A histochemical assay was routinely performed of myoadenylate deaminase (MAD) in muscle biopsy specimens.
      
The biopsy specimens in all of these patients were essentially normal and in none of them was the diagnosis of a neuromuscular disease made.
      
The presence of circulating anti-peripheral nerve antibodies and of immunoglobulin deposits in nerve biopsy specimens was investigated by immunofluorescence techniques but failed to demonstrate any immunoreactivity.
      
In order to investigate the pathological basis of muscle hypertrophy in Duchenne dystrophy, 9 biopsy specimens of the lateral gastrocnemius and 7 of the vastus lateralis were compared.
      
Histochemical and biochemical studies showed a lack of G6PD activity in muscle biopsy specimens as well as in erythrocytes.
      
Creatine kinase isoenzymes were determined in skeletal muscle biopsy specimens of 34 patients suffering from neurogenic muscular atrophies.
      
Histological and ultrastructural studies were performed on nerve and muscle biopsy specimens from two patients with the syndrome of continuous muscle fibre activity.
      
 

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