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renal cells
We studied certain aspects of interaction between (3H)aldosterone and the cytoplasmic as well as nuclear receptors of renal cells in the rats during compensatory renal hypertrophy at the background of reflex renal dystrophy.
      
Angiotensin II (ANG II) has multiple effects on cardiovascular and renal cells, including vasoconstriction, cell growth, induction of proinflammatory cytokines, and profibrogenic actions.
      
ANG II-induced ROS play a pivotal role in several pathophysiologic situations of vascular and renal cells such as hypertension, endothelial dysfunction, nitrate tolerance, atherosclerosis, and cellular remodeling.
      
These findings suggest that pHGF may non-specifically promote the DNA synthesis of renal cells.
      
The results suggested that NaF induces the apoptosis in renal tubules via activation of the Bax expression and Bcl-2 suppression; OPN probably acts as protective role against apoptosis in fluoride-treated renal cells.
      
In the present study, the effect of fluoride on intracellular free calcium ([Ca2+]i) and Ca2+-ATPase of renal cells were examined.
      
Quantification of dome formation by image analysis in LLC-PK1 renal cells: A sensitive method for the assessment of in vitro nep
      
Isoforms of antiporter isolated from oocytes (XL-NHE) and renal cells of the clawed frog Xenopus laevis (XNHE) have also been described.
      
Since renal cells in culture release KHCO3 in response to hypotonic stress [11], we investigated the effect of an acute water load on urinary KHCO3 excretion in 5 healthy individuals.
      
TGF-β1 impairs homocysteine metabolism in human renal cells: possible implications for transplantation
      
Different rates of cellular uptake also may play a role in time course of toxicity of the cysteine conjugates and the mercapturic acids in the renal cells.
      
In addition, it is also possible that different intracellular interactions in renal cells play a role in protection against the nephrotoxicity of high doses of CDDP.
      
Pretreatment of isolated renal cells with pertussis toxin (PTX, 500 ng/ml) overnight did not attenuate but significantly increased ET-1-induced IP-formation.
      
Thus, the appearance of cellular fragments represents acute damage to renal cells caused by various means.
      
On the contrary, its presence on renal cells may act to mediate bacterial colonization.
      
The malignant renal cells were treated with different cytokines (GM-CSF, G-CSF and interleukin-3) in different dosages.
      
Primary cultures of polarized proximal tubular epithelial cells are useful then as an in vitro model to study the crystallizing activities of polyanionic macromolecules produced by renal cells.
      
Increased calcium oxalate crystal nucleation and aggregation by peroxidized protein of human kidney stone matrix and renal cells
      
The present data suggest that acute exposure to a high concentration of oxalate challenges the renal cells, diminishes their viability and induces changes in cytosolic Ca2+ levels.
      
The effect of magnesium and calcium on the efficiency of calcium oxalate crystal binding to renal cells was also investigated.
      
 

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