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myocardial fibrosis
Midwall myocardial fibrosis in Becker-Kiener muscular dystrophy
      
In chronic myocarditis, viremia is often absent and myocardial fibrosis and dilated cardiomyopathy (DCM) resulting in heart failure can occur.
      
MRI is a sensitive tool detecting myocardial fibrosis on late images after application of paramagnetic contrast agents.
      
Patchy or homogenous myocardial fibrosis in Becker muscular dystrophy
      
Transforming growth factor-β receptor antagonism attenuates myocardial fibrosis in mice with cardiac-restricted overexpression o
      
The mechanisms that are responsible for the development of myocardial fibrosis in inflammatory cardiomyopathy are unknown.
      
The MHCsTNF mice develop a heart failure phenotype that is characterized by progressive myocardial fibrosis, as well as increased levels transforming growth factor-β (TGF-β)(mRNA and protein.
      
In 80% of patients sudden death is due to myocardial ischemia or interstitial myocardial fibrosis which in turn result from acquired diseases such as coronary artery disease and hypertension.
      
Aldosterone and Myocardial Fibrosis in Heart Failure
      
Areas showing LGE correspond to zones of myocyte necrosis or myocardial fibrosis as shown by comparison with histopathology.
      
Effects of ligustrazine on myocardial fibrosis in rats with pressure overload
      
Objective: To investigate the effects of ligustrazine (Li) on myocardial fibrosis in rats with pressure overload.Methods: Pressure overload rat models were established by constricting the abdominal aorta.
      
In this review we examine the recent literature on myocardial fibrosis and myofibroblast activity, highlighting the effects of several classes of cardiovascular agents on the remodeling process.
      
The most exciting development is the application of late gadolinium-enhanced (LGE) imaging, which allows for in vivo detection of myocardial fibrosis.
      
The most exciting development is the application of late gadolinium-enhanced (LGE) imaging, which allows for in vivo detection of myocardial fibrosis.
      
Aldosterone produces adverse effects on the vasculature (endothelial dysfunction) and on the myocardium (myocardial fibrosis).
      
The presence of angiotensin-converting enzyme (ACE) activity and angiotensin II receptors was examined in relation to myocardial fibrosis.
      
Aldosterone is an important downstream mediator of the reninangiotensin-aldosterone system that promotes myocardial collagen deposition, myocardial fibrosis, apoptosis, ventricular remodeling, and endothelial dysfunction.
      
1), including enhanced potassium and magnesium excretion, myocardial fibrosis, inhibition of catecholamine reuptake, endothelial cell and baroreceptor dysfunction, and ventricular arrhythmias.
      
In humans and in rat models, aldosterone action induces heart remodeling and interstitial and perivascular myocardial fibrosis.
      
 

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