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prostatic hypertrophy
Further investigations revealed as hydronephrosis secondary to a benign prostatic hypertrophy as the cause of acute renal failure
      
The gold standard for definitive management of symptomatic benign prostatic hypertrophy is transurethral resection of the prostate (TURP).
      
The gold standard for definitive management of symptomatic benign prostatic hypertrophy is transurethral resection of the prostate (TURP).
      
Each fungus can cause changes in the prostate that mimic bacterial infection, benign prostatic hypertrophy, or neoplasm.
      
Nocturia, aging, benign prostatic hypertrophy, and nocturnal vasopressin
      
Curative effect of electro-acupuncture and Tuina on prostatic hypertrophy
      
Objective To observe the curative effect of electro-acupuncture plus Tuina on prostatic hypertrophy.Method Fifty-six cases of prostatic hypertrophy were treated according to the principle of taking the kidney as the main aspect.
      
α1-adrenoceptor subtype affinities of drugs for the treatment of prostatic hypertrophy
      
A systematic study of testosterone metabolism in benign prostatic hypertrophy (BPH): In vitro results
      
Forty patients undergoing prostatectomy for benign prostatic hypertrophy were included in a double blind trial of epsilon aminocaproic acid, and the incidence of postoperative deep vein thrombosis determined, using the 125I-fibrinogen technique.
      
10 patients aged 31 to 72 years were treated with ethinyl-norgestrienone with different schedules and 3 men suffering from benign prostatic hypertrophy were treated with medroxyprogesterone acetate.
      
The determination of steroid-receptors in human prostatic tissue from 68 patients with benign prostatic hypertrophy(BPH) and 36 patients with prostatic cancer by the method of cold agargel-electrophoresis is described.
      
2) The incorporation of labelled T into minced benign prostatic hypertrophy (BPH) tissue from subjects with high (40 ng/ml) hPr was measured and was found to be more than twice the uptake into tissue from those with low hPr (6.5±1.9 ng/ml).
      
Tissue levels of free monosubstituted guanidines in human benign prostatic hypertrophy
      
Homogenates of tissue of benign prostatic hypertrophy have been analysed by means of ion exchange chromatography for their content of free monosubstituted guanidines.
      
In patients with benign prostatic hypertrophy treated before prostatectomy with oestrogens or antiandrogens the lactoferrin concentration is decreased.
      
The corresponding values for patients with benign prostatic hypertrophy (BHP) and carcinoma of the prostate (Ca) were 297.8±44.7 and 1592.1±622.7 respectively.
      
The "irritative" symptoms of prostatic hypertrophy, including nocturnal frequency and dysuria, were improved after Nicergoline significantly more than after placebo.
      
In 10 further patients with prostatic hypertrophy, peak and mean flow rates increased by 50% and 77% respectively after the acute administration of Nicergoline.
      
Prostatic hypertrophy and venereal disease as possible risk factors in the development of bladder cancer
      
 

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