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retinoic acid receptor
The nuclear retinoic acid receptor beta 2 (RAR-β2) is supposed to be a prognostic marker of retinoid sensitivity in patients after retinoid treatment.
      
PPARα activated by fibric acids form heterodimers with the 9-cis retinoic acid receptor (RXR).
      
Acute promyelocytic leukemia (APL) is a disease associated with fusion oncoproteins invariably involving the retinoic acid receptor (Rarα).
      
Acute promyelocytic leukemia (APL) is characterized by a translocation between the promyelocytic leukemia gene (PML) on chromosome 15 and the retinoic acid receptor-α (RARα) gene on chromosome 17.
      
Acute promyelocytic leukemia (APL), characterized by a translocation between the promyelocytic leukemia gene (PML) on chromosome 15 and the retinoic acid receptor-α (RARα) gene on chromosome 17, has become a model for targeted treatment of cancer.
      
The present study was undertaken to investigate the effects of selenite (SeIV) and selenate (SeVI) on the all-trans retinoic acid (RA)-nuclear retinoic acid receptor (RAR) complex formation in rat liver.
      
PPARs heterodimerize with 9-cis retinoic acid receptor (RXR), and bind to PP response element(s) (PPREs) on the target gene promoter to initiate inducible transcriptional activity.
      
Retinoic acids bind to the retinoic acid receptor (RARα) component of the fusion product, resulting in degradation of the fusion protein by ubiqutinization.
      
Deficiency of TH during development results in severe cerebral abnormalities similar to those seen in the mouse when the retinoic acid receptor (ROR)α gene is disrupted.
      
Acute promyelocytic leukemia (APL) is a human cancer generated by a chromosomal translocation t(15;17) involving the promyelocytic leukemia (PML) and retinoic acid receptor alpha (RARα) genes.
      
Retinoic acid receptor α and retinoid X receptor specific agonists reduce renal injury in established chronic glomerulonephritis
      
We describe a patient with promyelocytic leukemia zinc finger/retinoic acid receptor alpha (PLZF/RARα) APML who was treated with combination chemotherapy after poor response to arsenic trioxide.
      
The acute promyelocytic leukemia-specific PML-RARα fusion protein is a dominant-negative transcriptional repressor of retinoic acid receptor (RAR) target genes, which recruits HDAC and corepressor proteins and inhibits coactivators.
      
Hyperacetylation enhances the growth-inhibitory effect of all-trans retinoic acid by the restoration of retinoic acid receptor β
      
Patterns of expression of retinoic acid receptor beta 2 (RAR-β2)-LacZ reporter gene in the embryonic foregut
      
Our aim was to study the pattern of expression of retinoic acid receptor beta 2 (RAR-β2) in the region of the foregut during the early stages of embryonic development.
      
Immunochemical and in situ hybridization analyses of retinoic acid receptor α, β, and γ in murine Harderian and submandibular gl
      
Expression of a dominant negative retinoic acid receptor γ in Xenopus embryos leads to partial resistance to retinoic acid
      
Expression and function of a retinoic acid receptor in budding ascidians
      
We isolated a cDNA clone from this species, named PmRAR, encoding a retinoic acid receptor (RAR) homologue.
      
 

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