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The aim of this paper was to investigate the change of serum leptin and its relationship with platelet membrane glycoprotein Ib (GP Ib) in patients with coronary heart disease (CHD).
      
The enrolled included 50 patients with CHD (CHD group) and 30 patients without CHD (control group) who were diagnosed by coronary angiography.
      
The positive percentage and the average fluorescence intensity of platelet membrane GP Ib in the CHD group were significantly lower than those in the control group (P >amp;lt; 0.05).
      
Single factor correlative analysis revealed that serum leptin in CHD patients was negatively correlated with the average fluorescence intensity of platelet membrane GP Ib (P >amp;lt; 0.05).
      
Multifactorial stepwise regression analysis showed that serum leptin in CHD patients was independently negatively correlated with the average fluorescence intensity of platelet membrane GP Ib (P >amp;lt; 0.05).
      
Logistic analysis demonstrated that serum leptin was independently correlated with the risk of CHD (P >amp;lt; 0.05).
      
Hyperleptinemia may play an important role in the pathogenesis of CHD.
      
The inotropic response of isolated myocardial strips to α1-adrenoceptor stimulation was compared for patients with chronic coronary heart disease (CHD) and patients with WPW syndrome.
      
The myocardium of the CHD patients was characterized by a three-phase response.
      
The specific features of the inotropic response to α1-adrenoceptor stimulation in the CHD patients were assumed to be determined by changes in intracellular homeostasis of Ca2+.
      
Electromechanical coupling in cardiac myocytes of CHD patients depends on Ca2+ deposited in the sarcoplasmic reticulum to a greater extent than coupling in the intact myocardium.
      
The effects of Amiodarone and Nibentan on the postextrasystolic and postrest reactions were studied with normal rat myocardium and (for Amiodarone only) with myocardial strips isolated from patients with coronary heart disease (CHD).
      
It was supposed that, in the myocardium of some CHD patients (as well as in the rat myocardium), Amiodarone increases the ability of the sarcoplasmic reticulum to accumulate calcium ions.
      
Two groups of volunteers participated in the tests: a control group of 32 healthy subjects (group 1) and a group of 35 coronary heart disease (CHD) patients (group 2).
      
However, the groups of healthy subjects and CHD patients differed in the reactivity of indices, the amplitude of changes in nonlinear indices being narrower in the latter group than in group 1.
      
The influence of excessive body mass on the development of cardiorespiratory deficiency was studied in patients with coronary heart disease (CHD).
      
In the CHD patients with excessive body mass, mean blood pressure, cardiac output, left ventricular power output, and left ventricular posterior wall thickness were significantly increased even at rest.
      
The relationship between the carbon dioxide ventilatory equivalent and hemodynamic parameters during exercise was studied in healthy subjects and coronary heart disease (CHD) patients.
      
Gas exchange, ventilatory control, and central hemodynamics during graded exercise were analyzed in 85 subjects, including 32 healthy subjects and 53 CHD patients.
      
Twenty-seven CHD patients had coronary insufficiency but not heart failure and had a left ventricle ejection fraction of ≥50%; 26 patients had chronic heart failure and an ejection fraction of >amp;lt;40%.
      
 

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