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fibrogenesis
The liver fibrogenesis was assessed using a digital image analysis instrument of Masson's trichrome stained sections.
      
It is associated with increased fibrogenesis and, in nongenotype 3 infection, with a reduced response to antiviral therapy.
      
Great progress has been made over the past 20 years in elucidating the mechanisms of hepatic fibrogenesis.
      
The recognition of the hepatic stellate cell as the fibrogenic cell of the liver, as well as the recognition of key cytokines involved in fibrogenesis, has facilitated the development of targeted antifibrotic therapies.
      
Great progress has been made over the past 20 years in elucidating the mechanisms of hepatic fibrogenesis.
      
The recognition of the hepatic stellate cell as the fibrogenic cell of the liver, as well as the recognition of key cytokines involved in fibrogenesis, has facilitated the development of targeted Antifibrotic therapies.
      
There is a clinical need for noninvasive measurement of liver fibrosis both to diagnose significant liver fibrosis and to monitor the effects of therapy on fibrogenesis and fibrolysis.
      
Whether metabolic steatosis has a direct role in liver fibrosis progression or is only a surrogate marker of an underlying defect triggering fibrogenesis, such as insulin resistance, is a subject of debate.
      
Exciting new data are expanding our understanding of the mechanisms of steatogenesis in HCV infection and providing potential links between insulin resistance or hyperglycemic states and liver fibrogenesis.
      
Future treatment for patients failing to achieve weight loss goals is likely to consist of combination therapy targeting insulin resistance, oxidative stress, and fibrogenesis.
      
This article reviews our current understanding of the origins of mesenchymal cells and their role in excessive scarring and fibrogenesis and in the systemic fibrotic disease scleroderma.
      
The expression of basic fibroblast growth factor (bFGF) in rat liver fibrosis and hepatic stellate cells (HSCs) and the relationship between the expression of bFGF and rat liver fibrogenesis were studied.
      
It is suggested that bFGF is related with liver fibrosis of rats closely and may be a fibrogenesis factor of liver.
      
bFGF possibly regulates liver fibrogenesis through regulating metabolism of extracellular matrix (ECM) by autocrine and paracrine stimulation.
      
N-terminal procollagen-III peptide (P-III-P) has been considered a marker of fibrogenesis and inflammatory activity of the liver.
      
This chronic injury leads to changes in the cellular property of the liver and subsequently causes fibrogenesis to demolish normal liver blood system.
      
TGF-β1 is a key factor in fibrogenesis, and the angiotensin?II receptor antagonist losartan may decrease the intra-graft synthesis of TGF-β1.
      
Fibrogenesis imperfecta ossium: imaging correlation in three new patients
      
Fibrogenesis imperfecta ossium is an extremely rare disorder that can easily be misdiagnosed.
      
We retrospectively reviewed the clinical and imaging data of three confirmed cases of fibrogenesis imperfecta.
      
 

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