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bladder tumor
A suspension of heat-killed Aspergillus fumigatus mycelium inhibited the growth of a chemically-induced mouse bladder tumor (MBT).
      
Lymphocytes from patients with transitional cell carcinoma (TCC) of the urinary bladder are more cytotoxic to bladder tumor cells than to a variety of control cells.
      
Induction of TH1- and TH2-associated cytokine mRNA in mouse bladder following intravesical growth of the murine bladder tumor MB
      
Large cooperative trials are more likely than series studied by small groups to bring about significant progress in the field of intravesical adjuvant chemotherapy of superficial bladder tumor
      
The purpose of this trial was to minimize local infalammation caused by intravesical instillation of antitumor agents, especially Adriamycin, in the treatment of bladder tumor.
      
Effects of N-trifluoroacetyladriamycin-14-valerate (AD-32) on human bladder tumor cell lines
      
We have compared the in vitro activity of N-trifluoroacetyladriamycin-14-valerate (AD-32) and doxorubicin hydrochloride (ADR) on the clonal growth of human bladder tumor cell lines (HBTCL).
      
The clinical results obtained in 81 cases of bladder tumor are used as a basis for discussion of factors influencing the effect of bladder instillation therapy.
      
From October 1983 to September 1985, 84 patients with superficial bladder tumor (Ta, Tl, Tis) were treated with sequential instillation of mitomycin C (MMC) and adriamycin (ADM).
      
Pretreatment with bismuth nitrate depressed the renal toxicity of CDDP without compromising its activity against a transplantable human bladder tumor.
      
In this study the antimicrotubular agents taxol, taxotere, and vinblastine were compared for their ability to inhibit the clonal growth of human bladder tumor cell lines using a soft-agar clonogenic assay.
      
This study was performed in human bladder tumor histocultures treated with several drugs (doxorubicin, mitomycin C, paclitaxel and 5-fluorouridine).
      
Bladder tumor induction by cytotoxic agents clinical experience and experimental data
      
In this paper, we will review the mechanism of action of sensitizers and show that, against the MBT-2 mouse bladder tumor, a number of sensitizing agents increased the antitumor activity of cyclophosphamide.
      
FCM was applied to 400 patients (225 without a urinary tumor and 175 with a past or recent bladder tumor).
      
The use of this technique must be more selective in bladder tumor evaluation.
      
Urine of 192 patients with different types, grades, and stages of bladder tumor and 72 controls were evaluated using dot-EIA.
      
Pathologic analysis of the bladder tumor and regional lymph nodes will help direct the need for adjuvant therapy in high-risk individuals.
      
Furthermore, the level of maspin was correlated to the growth rate of bladder tumor cell lines in vitro and in vivo.
      
Moreover, in pre-clinical bladder tumor models, inhibition of Survivin expression and/or function has been shown to impede tumor cell proliferation, and markedly induce spontaneous or chemotherapy induced apoptosis.
      
 

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