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postmortem
This accelerated atherosclerosis has also been recognized at postmortem examinations.
      
The symptoms, signs, results of X-ray examination after poisoning, as well as detailed postmortem pathological examination and determination of mercury content in different organs were presented.
      
To determining the postmortem interval (PMI) through quantitative analysis of the DNA degradation of cell nucleus in human brain and spleen by using image analysis technique (IAT).
      
The changes of retinal nuclear DNA content in rats after death was detected and the relationship between degradation of retinal nuclear DNA and postmortem interval (PMI) was analyzed.
      
A correlation between the postmortem interval (PMI) and AG was identified and the corresponding regression equation was obtained.
      
Gross and microscopic ocular findings were prospectively studied in 38 human immunodeficiency virus (HIV)-seropositive subjects undergoing postmortem examination.
      
Relative postmortem stability of spinal motoneuronal proteins detectable by two-dimensional electrophoresis
      
It was found that many of the proteins of bovine spinal motoneurons detectable on two-dimensional polyacrylamide gels appear to be relatively stablein situ at room temperature during the first postmortem day.
      
Upon visual inspection of the gels, postmortem changes in the amount of stain associated with a spot were obvious in three of 364 proteins from isolated motoneuronal cell bodies and none of 237 proteins from ventral roots.
      
In the neuropil surrounding the motoneuron cell bodies, more pronounced changes in protein patterns occurred during the postmortem period.
      
We conclude that properly controlled two-dimensional electrophoretic analyses of postmortem spinal tissue can provide reliable qualitative and quantitative information about the antemortem protein composition of spinal motoneurons.
      
Agonal status affects the metabolic activity of nerve endings isolated from postmortem human brain
      
Isolated nerve endings (synaptosomes) that show high rates of metabolic activity have been prepared up to 24 h postmortem from the brains of patients who have died suddenly.
      
Because of the presence of somatostatin-like immunoreactivity (SLI) in limbic system nuclei of the brain, we examined postmortem concentrations of SLI in patients dying with schizophrenia and in normal controls.
      
The blockage of electrical conduction was monitored by recording the amplitudes of evoked potentials during administration of the damaging substances, and damage was assessed, by postmortem histological examination.
      
The effect of postmortem delay on the distribution of microtubule-associated proteins τ, MAP2, and MAP5 in the rat
      
Studies of such diseases in the human involve the use of postmortem brain tissue.
      
Postmortem delay may vary considerably from a few hours to a few days, and within this period, a degree of cytoskeletal breakdown may occur.
      
It is therefore crucial to examine alterations occurring in the cytoskeleton as a result of postmortem delay and subtract these from those caused by the disease.
      
In this study, the distribution of τ, MAP2, and MAP5 immunohistochemistry was examined following postmortem intervals of 0-72 h in the rat cerebral cortex, corpus callosum, caudate nucleus, and hippocampus.
      
 

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