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Each microtubule-associated protein (MAP) underwent unique changes that were dependent both on postmortem interval and the brain region examined.
      
Following long postmortem delays, some of the changes in these proteins were similar to those seen in rodent models of cerebral ischemia.
      
These results demonstrate that MAPs are not stable during postmortem delay in the rat.
      
Therefore, caution must be exercised when interpreting changes in MAPs in human postmortem tissue, especially in cases where ischemic injury may be involved.
      
Examination of control tissue carefully matched for postmortem delay is therefore essential to allow meaningful interpretation of cytoskeletal abnormalities in human neurodegenerative disease.
      
Brain regions from postmortem controls and HD- or AD-affected individuals were assayed to determine the activity of NAALADase as well as the levels of NAAG,N-acetylaspartate (NAA), and several amino acids.
      
We describe a complementary immunoblotting approach, for the assessment of neuronal cytoskeletal proteins, which employs fresh frozen postmortem tissues.
      
This study suggests that data obtained from treating rats with antipsychotic drugs cannot be simplistically extrapolated to studies on tissue obtained postmortem from schizophrenic subjects treated with the same drugs.
      
The gross and microscopic ocular findings were prospectively studied in 38 human immunodeficiency virus (HIV)-seropositive subjects undergoing postmortem examination.
      
Concentrations of zinc in postmortem serum and four brain regions were measured by flame atomic absorption spectrometry and instrumental neutron activation analysis, respectively, in nine Alzheimer's disease (AD) and eight control subjects.
      
The uterine tubes were resected postmortem or surgically removed from patients with uterine myoma.
      
In this work, we studied the processing of proenkephalin-derived peptides in postmortem human pituitary (PMHP), ACTH-producing adenomas (ACTH-PA), nonfunctioning adenomas (NFA), and GH-producing adenomas (GH-PA).
      
No clinical imaging method can come close to meeting the first requirement, leading to the disadvantage of being limited to postmortem studies.
      
To predict the results of postmortem brain examinations, we applied ANNs to the Nun Study data set, a longitudinal epidemiological study, which includes annual cognitive and functional evaluation.
      
In an unselected consecutive postmortem series, 53.2% of the adrenals showed cortical nodules with diameters up to 1 mm in 13%, between 1 and 2 mm in 28% and >amp;gt;2 mm in 12%.
      
Fat-cell metaplasia in the adrenal cortex: Incidence, structure, and correlation to basic diseases in a postmortem series
      
In a postmortem series of adrenals from 497 patients 25 cases (5%) showed foci of fat-cell metaplasia or bone-marrow metaplasia in the zona fasciculata or the zona reticularis.
      
Recent studies have shown a higher incidence of C-cell hyperplasia (CCH) in men compared with women in postmortem thyroid tissues.
      
AR protein was detected in most surgically resected thyroids with MTC and CCH (80%), but in only 25% of autopsy thyroids, probably reflecting postmortem degradation of the receptor protein.
      
These results support the observation that CCH is more common in postmortem thyroids of males and suggest that the presence of AR with higher circulating levels of androgens may contribute to the higher incidence of CCH in men.
      
 

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