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penile erection
Intermittent penile erection in lumbar canal stenosis
      
Penile erection is a neurovascular event modulated by psyche and hormones.
      
Erectile dysfunction (ED) has been classified as psychogenic, arteriogenic, neurogenic, endocrinologic, and cavernosal, based on the organs that are involved in penile erection.
      
ED in diabetes results from combinations of impairments from nearly every step in the production of a penile erection.
      
Phosphodiesterase (PDE) 5 inhibitors reduce cyclic guanylate monophosphate breakdown, promoting vascular relaxation in the corpora cavernosa and penile erection during sexual stimulation.
      
Priapism is defined as a pathologic condition in which penile erection persists beyond, or is unrelated to, sexual stimulation.
      
Erectile dysfunction (ED), defined as the consistent inability to obtain and/or maintain penile erection sufficient for satisfactory sexual relations, is associated with a variety of medical, psychological, and lifestyle risk factors.
      
We hypothesize that the primary action of nitric oxide and other agents that cause penile erection is inhibition of the RhoA/Rho-kinase pathway, thereby allowing vasodilation and erection.
      
Penile erection occurs in response to visual, olfactory, imaginative, and tactile stimuli initiated within the brain and/ or on the periphery.
      
Erectile dysfunction, defined as the consistent inability to obtain and/or maintain a penile erection sufficient for adequate sexual relations, also is a common problem.
      
In this article, functional anatomy and physiology of human penile erection is reviewed, as are current clinical vasoactive agents including prostaglandin E-1, papaverine, and phentolamine.
      
We examined the effects of trans-resveratrol on male reproductive functions; ex-vivo penile erection and in-vivo sperm counts and quality.
      
In conclusion, we propose that resveratrol has a positive effect on male reproductive function by triggering a penile erection, as well as enhancing blood testosterone levels, testicular sperm counts, and epididymal sperm motility.
      
Role of nitric oxide in penile erection and yawning induced by 5-HT1c receptor agonists in male rats
      
m-CPP- and TFMPP-induced penile erection and yawning was prevented also by the i.c.v.
      
The results suggest that central nitric oxide is involved in the expression of penile erection and yawning induced by 5-HT1c receptor agonists.
      
Prevention by morphine of apomorphine- and oxytocin-induced penile erection and yawning: involvement of nitric oxide
      
Either apomorphine (80 μg/kgs.c.) or oxytocin (30 ng i.c.v.) increased significantly basal NO2- and NO3- concentration in the paraventricular dialysate, penile erection and yawning.
      
A slight to moderate increase in degree of penile erection was also observed for 6 of 8 patients, but the erection ability was insufficient to consummate sexual intercourse.
      
Lisuride increased the incidence of stretching and yawning (SY) as well as of penile erection (PE) and elicited stereotyped behavior (SB), aggressive behavior and mounting in male rats, depending on the dose used.
      
 

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