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mammary cancer
We investigated the molecular mechanism of Lovastatin anti-tumor function through the study of its effect on membrane ion flow, gap junctional intercellular communication (GJIC), and the pathways of related signals in MCF-7 mammary cancer cells.
      
Objective: To determine the effect of cis-9, trans-1 1-conjugated linoleic acid on the cell cycle of mammary cancer cells (MCF-7) and the possible mechanism of the inhibitory effect of c9,t11-CLA.
      
Therefore, it could be regarded as an effective remedy for treatment of HDB and prevention of mammary cancer genesis.
      
Clinical study on treatment of mammary cancer by Shenqi Fuzheng Injection (参芪 扶正注射液) in cooperation with chemotherapy
      
Reversal of adriamycin resistance in human mammary cancer cells by small interfering RNA of MDR1 and MDR3 genes
      
Molybdenum (Mo) supplementation reduces the incidence of nitrosamine-induced tumors in the esophagus and forestomach of laboratory animals, and the incidence of mammary cancer in female rats induced byN-nitroso-N-methylurea (NMU).
      
I have used the paradigm of mammary cancer induction by the mouse mammary tumor virus (MMTV) to illustrate the body of evidence that supports the hypothesis that mammary epithelial stem/progenitor cells represent targets for oncogenic transformation.
      
In a prospectively planned and randomised study 7 clinically active anti-cancer agents were tested as single-agents and in double-combinations by simultaneous application against the autochthonous mammary cancer of the rat.
      
Remission rates similar to those obtained in the mammary cancer of the woman could not be obtained at the dosages and schedules chosen.
      
Mouse mammary cancer (Shionogi carcinoma-115) has been extensively used to analyze the mechanism of androgen-dependent cancer growth.
      
Expression of complement protein C5a in a murine mammary cancer model: tumor regression by interference with the cell cycle
      
In DMBA-induced mammary cancer in rats Fc-1157a decreased the number of new tumors and inhibited the growth of existing tumors, these effects being statistically highly significant.
      
FdUrd at doses in the range 5-100?μg/animal was effective against meningeal carcinomatosis using Walker 256 carcinoma cells in rats and MM46 mammary cancer cells in mice and against meningeal gliomatosis using 203 glioma cells in mice.
      
In ten rats, a small piece of chemically induced mammary cancer was transplanted to the supraoccipital bone.
      
The large percentage of female patients is due to the predominant role of mammary cancer (50%) and the comparatively long survival of patients after a primary diagnosis of this type of carcinoma.
      
Combined surgical, radiotherapeutic, and sometimes chemotherapeutic treatment of early stages of operable mammary cancer results in 5-year survival without recurrence in a similar percentage of cases as modified radical mastectomy does.
      
Disorders in the metabolism of calcium of the breast are manifested by calcifications of the acini, ducts, epithelial cells and by necrosis and in patients with advanced mammary cancer by hypercalcemia.
      
1-, 2-, 3- and 9-Aminophenanthrenes produce mammary cancer with approximately the same activity when fed to female Sprague-Dawley rats.
      
The incidence of mammary cancer was significantly increased by postnatal applications of cyclophosphamide or Freunds adjuvant.
      
Progesterone receptors in feline mammary cancer cytosol
      
 

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