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icr mice
Twenty-five tiptoe-walking Yoshimura (twy) mice with calcified mass lesions compressing the spinal cord posterolaterally at the C1-C2 vertebral levels were compared with five Institute of Cancer Research (ICR) mice that served as controls.
      
albicans) in mice, the fungal vaginitis model was established in female ICR mice by intravaginal inoculation of suspension of C.
      
To study the effect of itraconazole on the vaginal candidiasis caused by Candida under different immunity conditions, the fungal vaginitis model was established in female ICR mice by intravaginal inoculation of suspension of C.
      
The relationship between Ni-induced hepatic lipid peroxidation (LPO) and the concentrations of Ni and trace elements was investigated in male ICR mice.
      
The l-SeMet α,γ-elimination enzyme was ubiquitous in tissues from ICR mice and the activity was relatively high in the large intestine, brain, and muscle, as well as the liver.
      
Isolated islets from ICR mice were transplanted into the renal subcapsular space of streptozotocin-induced diabetic C57BL/6 mice.
      
Cocaine HCl (0, 10, or 50 mg/kg) was injected into adult male ICR mice ip.
      
ICR mice treated once with LC 1 day or 2 days before challenge survived lethal infection, but untreated orLactobacillus fermentum (LF)-treated mice did not.
      
Experimental induction of various kind of tumours in ICR mice with cigarette tar
      
Induction of tumors in ICR mice with N-nitrosopiperidine, especially in forestomach
      
Data from 1,919 outbred ICR mice were used to examine the potential usefulness of growth curve parameters as selection criteria for altering the relationship between body weight and age.
      
Replicated divergent selection was conducted for two generations in ICR mice for in vitro developmental capacity (IVDC; percentage of fertilized one-cell zygotes developing to blastocysts in vitro per female donor).
      
GTG obesity caused diabetic changes likewise in ICR mice as manifested by hyperglycemia, glucosuria, hyperinsulinemia, and depressed insulin sensitivity.
      
In vitro glucose oxidation was also decreased to about half of the untreated controls and became less sensitive to added insulin in the epididymal adipose tissue from GTG-obese ICR mice.
      
By contrast, either hyperglycemia or glucosuria did not develop in GTG-obese C57BL mice, although the other diabetic changes observed in GTG-obese ICR mice were induced but only in lesser extents.
      
Parabiosis with normal control ICR mice for 2 weeks decreased the blood glucose level to 260±51 mg/ 100ml (P>amp;lt;0.01) and resulted in serum insulin levels of 46.0±18.0 μU/ml (P>amp;lt;0.01).
      
Epinephrine-induced hyperglycaemia and isoproterenol-induced hyperinsulinaemia, both of which could be blocked by β-adrenergic antagonists, were more marked in KK mice than in ICR mice.
      
The cell-mediated cytotoxicity to islet cells of non-obese diabetic mice was significantly higher than that of control ICR mice.
      
In contrast, natural killer and antibody-dependent cell-mediated cytotoxicity activities of the spleen cells from the non-obese diabetic mice were significantly lower than those of ICR mice spleen cells.
      
The NSY (Nagoya-Shibata-Yasuda) mouse was established as an inbred strain of mouse with spontaneous development of diabetes mellitus, by selective breeding for glucose intolerance from outbred Jcl∶ICR mice.
      
 

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