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pancreatic
Inhibitive Effect of Prodigiosin on the Proliferation of Human Malignant Pancreatic Cancer Cells
      
Pancreatic cancer is not only common, but also extremely difficult to treat, for which it has been called "the challenge of the twenty-first century".
      
In this study, we find that prodigiosin could effectively inhibit the proliferation of human pancreatic cancer cells H8898 in a dose-and-time-dependent manner, with an IC50 of 75μmol according to the results of MTT and cell proliferation assays.
      
Prodigiosin also could induce apoptosis of pancreatic cancer cells at low concentration and results in the fragmentation pattern of DNA.
      
All these results demonstrate that prodigiosin can obviously inhibit the proliferation of pancreatic cancer cells H8898 by arresting the cell cycle and inducing apoptosis.
      
It is a cytosolic protein that protects cells from the adventitious invasion of pancreatic-type ribonuclease.
      
The rats in group A was subjected to retrograde injection of 5% sodium taurocholate via the pancreatic duct, and those in group B received chondroitin-sulfate therapy after ANP induction.
      
The morphologic changes in pancreatic tissues were observed.
      
Retrograde infusion of chondroitin-sulfate via pancreatic duct could alleviate the pancreatic cell damage as a sort of scavengers of oxygen free radicals.
      
Immunotherapeutic effects on murine pancreatic carcinoma by β-elemene combined with dendritic cells modified with genes encoding
      
Effect of end-to-end invagination pancreaticojejunostomy with circle discontinuous U suture in pancreatic surgery
      
The incidences of pancreatic fistula with different types of pancreaticojejunostomy were compared.
      
The overall incidence rate of pancreatic fistula was 26.5% (22/83).
      
No pancreatic fistula occurred in end-to-end invagination pancreaticojejunostomy with circle discontinuous U suture.
      
End-to-end invagination pancreaticojejunostomy with circle discontinuous U suture has a definite effect on avoiding pancreatic fistula following pancreaticojejunostomy and is worth being recommended.
      
Since first described in 1982, intraductal papillary mucinous neoplasm (IPMN) has been the preferred term to describe the proliferation of the pancreatic ductal epithelium.
      
It is totally different from pancreatic carcinoma in epidemiology, histology, pathology and prognosis.
      
A large branch duct IPMN and marked dilation of the main pancreatic duct indicate the presence of adenoma at least.
      
The average diameter of the pancreatic duct was also wider in the invasive group (8.7 mm vs 4.3 mm).
      
Significant predictors of malignant IPMNs included weight loss, jaundice, a high level of serum CA19-9, a large pancreatic duct and main-duct type carcinoma.
      
 

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