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renal carcinoma
Allele Deletion Mapping of the Short Arm of Human Chromosome 3 in Renal Carcinoma
      
Allelic deletions along the short arm of human chromosome 3 were mapped in 57 pairs of DNA samples from tumor and normal tissue of renal carcinoma patients in order to locate potential tumor suppressor genes.
      
VHL inactivation in sporadic clear cell renal carcinoma
      
Clear cell renal carcinoma (CCRC) accounts for 75% of all renal cancer cases.
      
Diagnosis and treatment of chromophobe cell renal carcinoma (report of 3 cases)
      
The aim of the present study was to investigate whether inhibition of telomerase activity by siRNA targeted against human telomerase RNA (hTR) can inhibit proliferation and induce apoptotic cell death in human renal carcinoma cells (HRCCs).
      
Our results suggest that As2O3 is probably a new candidate agent for the treatment of human renal carcinoma.
      
The role of resection for patients with renal carcinoma
      
Although cytotoxic chemotherapy continues to have a minor role in patients with clear cell renal carcinoma, it may become the treatment of choice for some patients with variant renal cancers.
      
Effects of peptide nucleic acids against Ki-67 gene on the proliferation and apoptosis of human renal carcinoma cell line
      
From these finds we are led to conclude that anti-sense PNAs targeting Ki-67 gene have stronger effects on the inhibition of the proliferation and induction of apoptosis of human renal carcinoma cells than ASODNs targeting Ki-67 gene.
      
Introduction: Standard therapy for recurrent or metastatic renal carcinoma includes the biologic response modifiers interferon-alpha (IFN-α) and interleukin-2 (IL-2).
      
Interferon-gamma (IFN-γ) is a type II interferon that demonstrated promising activity in renal carcinoma in early clinical trials.
      
In another hypercalcemic patient suffering from renal carcinoma, PTHrP measured by IRMA decreased by 40% within 12 h after nephrectomy, whereas PTHrP measured by RIA did not show a significant decline.
      
Measured by a monoclonal antibody based commercial enzyme-linked immunoassay, the Epo concentration was above the normal range, determined in nonanemic humans, in four of the renal carcinoma patients.
      
In addition, when monolayer cell cultures of 14 different established human renal carcinoma lines were screened, none of these released immunoreactive Epo in measurable amounts.
      
Vascularization of the chromophilic renal carcinomas was lower than that of the clear cell type of renal carcinoma.
      
Chemoimmunotherapy in the systemic treatment of advanced renal carcinoma
      
Early and late genetic changes in clear cell renal carcinoma
      
Renal transplantation in patients with bilateral renal carcinoma - how long should we wait
      
 

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