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hypothermia
The typical clinical features are hypothermia, bradycardia and neuropsychiatric manifestations such as lethargy.
      
In contract mild therapeutic hypothermia has been emphasized in the guidelines for post resuscitation care as one of the few major progresses improving prognosis.
      
The time of the onset of contracture was changed by preischemic infusion with buffer containing 0.5 mmoles/l iodoacetate (IAA) in order to get early contracture, and by hypothermia which delayed the development of contracture.
      
The first signs of contracture were noticed in normothermia (37°C) at 11 minutes, in hypothermia (26°C) at 25 minutes, and in the IAA-infused group at 3 minutes.
      
The myocardial perfusion-expressed as percent of myocardial area-was 99% at the precontracture state in normothermia, 95% in hypothermia and 100% in the IAA-infused group.
      
The effect of hypothermia during the period of calcium repletion on the calcium paradox
      
In this investigation the effect of hypothermia during reperfusion with calcium-containing solution on the calcium paradox damage in the isolated rat heart was studied.
      
The results show that hypothermia during the period of calcium repletion retards the sequence of events which ultimately results in release of large amounts of intracellular components.
      
Effects of nifedipine and low [Ca2+] on mechanical restitution during hypothermia in guinea pig papillary muscles
      
Hearts in Group I (n=8) were control without hypothermia, ischemia or pacing.
      
As compared to hypothermia alone, hypothermic cardioplegia offered a slightly better preservation.
      
But hypothermia alone or in combination with cardioplegia failed to completely normalize the post-ischemic R1F and PS values.
      
Regional hypothermia reduces myocardial necrosis even when instituted after the onset of ischemia
      
Mild Hypothermia reduces infarct size in the beating rabbit heart: A practical intervention for acute myocardial infarction
      
To further explore the potential of hypothermia for myocardial protection, rabbits underwent either a 30-, 45- or 60-min coronary artery occlusion and 3-h reperfusion.
      
Furthermore, to elucidate the mechanism of hypothermia's protection, we also examined its effect on isolated cardiomyocytes.
      
Hence blood pool cooling was easily induced and well tolerated and protected the beating heart against infarction even when hypothermia was started after the onset of coronary occlusion.
      
We conclude that hypothermia might be a simple and useful therapy for patients presenting with acute myocardial infarction.
      
Influence of mild hypothermia on myocardial contractility and circulatory function
      
Hypothermia increase isometric twitch force significantly by 91 ± 16% in human and by 50 ± 9% in pig myocardium (31 vs.
      
 

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