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hypothermia
In anesthetized pigs, mild hypothermia resulted in an increase in hemodynamic paramters of myocardial contractility.
      
Mild hypothermia exerts significant positive inotropic effects in human and porcine myocardium without increasing intracellular Ca2+-transients or SR Ca2+-content.
      
Hypothermia during reperfusion does not reduce myocardial infarct size in pigs
      
Intraventricular morphine produces pain relief, hypothermia, hyperglycaemia and increased prolactin and growth hormone levels in
      
The hypothermia in response to the injection of morphine was due to cutaneous vasodilation and sweating.
      
The results indicate that activation of opiate receptors in the brain produced pain relief, hypothermia (due to cutaneous vasodilation and sweating), and increased blood levels of prolactin, growth hormone and glucose in patients with cancer pain.
      
Baseline finger temperature (Tf) during hypothermia was significantly lower than during normothermia (Tf=32.4 ± 1.2 compared with 36.2 ±0.3°C, respectively), and than in the controls (Tf=34.8 ±0.8°C).
      
No significant differences in baseline skin blood flow and forearm blood flow were found between subjects during hypothermia or normothermia and controls, suggesting a failure of sympathetic drive to counter-regulate hypothermia in the subjects.
      
Skin vasoconstrictor responses to the contralateral cooling test and neck cooling test were markedly attenuated in three subjects, and to the finger cooling test in two subjects, during normothermia compared with hypothermia.
      
Blood presure responses to the Valsalva manoeuvre and head-up tilting were normal in all subjects, whereas the heart rate response to head-up tilting was blunted in three subjects during hypothermia.
      
Our findings during hypothermia and normothermia indicate that variations in core and skin temperature significantly affect skin vasomotor reactivity.
      
Various perfusion methods, regional or global hypothermia of at least 33 °C and pharmacological measures (fenoldopam, mannitol) to improve perfusion of the renal parenchyma are employed.
      
Clinical trials of potentially neuroprotective interventions such as hypothermia are under way.
      
Although medical management seems to fail in most of these patients, early hemicraniectomy and induced moderate hypothermia (32°C to 33°C) represent two novel therapeutic approaches to improve neurologic outcomes and decrease mortality rates.
      
Moreover, the recent favorable results of human clinical trials of hypothermia in human cardiac arrest and global brain ischemia have validated the general concept of neuroprotection for ischemic brain injury.
      
In refractory cases, hypothermia may be considered, although value of this strategy remains largely unexplored.
      
Moderate hypothermia (MH) is neuroprotective in animal models of focal ischemia when it is induced during, or within few hours after, onset of ischemia.
      
In patients with acute stroke, several observational studies suggested normothermia or mild hypothermia as independent prognostic factors for favorable outcome.
      
Currently, mild hypothermia was only examined in one clinical study that showed its feasibility and safety, but was not powered to examine efficacy.
      
Induced hypothermia: A novel neuroprotective treatment of neonatal encephalopathy after intrapartum hypoxia-ischemia
      
 

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