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sna
There was no obvious relationship between SNA and deaminase activity at higher levels of pH.
      
Schizosaccharomyces pombe replication protein Cdc45/Sna41 requires Hsk1/Cdc7 and Rad4/Cut5 for chromatin binding
      
With SNA, lactoferrin stained as a sharp main band at pI 5.1 with three additional weaker bands at IEPs from 4.8 to 4.9.
      
At this time a fine granular staining for SNA was visible at the sinusoidal sides of hepatocytes.
      
The hepatocytes of the outer limiting plate were specifically stained by WGA, Con A, and SNA in the 9th GW and the staining remained visible in developing bile ducts up to the 12th GW.
      
Therefore, PSL should be a superior reagent to the lectin from Sambucus nigra (SNA), which does not discriminate between α2,6-linked NeuAc5 present either in asparagine- or serine/threonine-linked oligosaccharides.
      
We observed a more restricted staining pattern by PSL as compared to SNA in paraffin sections from different rat organs.
      
In contrast, SNA staining was only partially sensitive to N-glycanase?F pretreatment demonstrating that it was in part due to α2,6-linked NeuAc5 present in serine/threonine-linked oligosaccharides.
      
The most striking observation in this regard was that PSL, in contrast to SNA, did not stain the mucus of sheep submandibular gland, which is extremely rich in serine/threonine-linked Neu5Acα2,6N-acetylgalactosamine.
      
Furthermore, in some tissues neuraminidase pretreatment resulted in increased intensity of SNA staining probably due to binding to exposed terminal N-acetylgalactosamine residues.
      
Ten lectins were used (ConA, WGA, PNA, SBA, DBA, LTA, UEA I, GSL II, MAL II and SNA) in combination with chemical and enzymatic treatments.
      
The curves were defined by calculating median blood pressure, SNA Range and reflex gain.
      
In both renal and cardiac sympathetics section of the carotid sinus and aortic nerves completely abolished the MAP-related changes in SNA.3.The renal baroreflex curves were reset from control levels during hypoxia.
      
Median blood pressure increased, as did SNA Range and gain.
      
Isolated perfusion of the lower brainstem with CO2-enriched solutions was performed and preganglionic sympathetic nerve activity (SNA) was recorded.
      
Perfusion of the RVLM with CO2enriched solutions over a period of 15 s induced a marked increase in SNA.
      
The magnitude of absolute changes in SNA during perfusion depended on the level of basal SNA before perfusion.
      
Microinjections of 4,4'-diisothiocyanatostilbene-2,2'-disulphonic acid (DIDS) and acetazolamide (ACZ) induced a marked rise in basal SNA, whereas diethylpyrocarbonate (DEPC) and ethylisopropylamiloride (EIPA) had no significant effect on basal SNA.
      
After application of DIDS and DEPC, the peak change in SNA due to perfusion of the RVLM with CO2-enriched solutions was slightly diminished.
      
Furthermore, neither ACZ nor EIPA produced any significant influence on the slope, peak change and time course of the increase in SNA compared with control perfusions.
      
 

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