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periodontitis
The different forms of periodontitis are all associated with a distinct bacterial flora.
      
However, a clear relationship is described between localized juvenile periodontitis (LJP) andActinobacillus actinomycetemcomitans.
      
Phagocytosis of Periodontopathogenic Bacteria by Crevicular Granulocytes Is Depressed in Progressive Periodontitis
      
Infections of the tissue surrounding the teeth (periodontitis) are usually caused by anaerobic gram-negative microorganisms.
      
Removal of these microorganisms can slow or arrest the progression of periodontitis.
      
Diabetes patients are at greater risk of developing periodontitis, may not respond as well to periodontal therapy as nondiabetic patients, and may require more aggressive treatment to manage periodontitis.
      
Microorganisms that cause periodontitis and the host response to these may increase insulin resistance in diabetic patients.
      
Treatment of periodontitis could improve glycemic control.
      
This may include appropriate treatment of moderate or severe periodontitis and of chronic bronchitis.
      
Host andPorphyromonas gingivalis proteinases in periodontitis: A biochemical model of infection and tissue destruction
      
Periodontitis is an excellent model of local tissue destruction due to the uncontrolled action of host and microbial proteinases.
      
In these cases, tooth extraction, removal of dental deposits, interrupted pulp treatment, apical periodontitis, or a relicted root were identified as causes of the development of erythema nodosum.
      
Our data indicate that the slow acetylator phenotype is associated with a higher risk of periodontitis, especially with respect to the severity of the disease.
      
Cloricromene, a coumarine derivative, reduced the development of periodontitis in rats
      
We investigated the effect of cloricromene in a rat model of periodontitis.
      
Periodontitis was induced in rats by placing a 2/0 braided silk ligature around the lower left first molar.
      
This suggests that cloricromene treatment, which reduced tumour necrosis factor production, may be of benefit in the treatment of periodontitis.
      
Fifty-one sites of periodontitis, 15 periodontal abscesses, 14 sites of dental caries with root canal infection, 23 sites of dental caries without root canal infection, and seven necrotic pulps were found by dental examination.
      
The relationship between periodontitis and pathogenesis of BD has not yet been determined.
      
Periodontitis-induced autoinflammatory response also may play a role in the development/severity of BD and RAS via IL-1 gene alteration.
      
 

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