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liver fibrosis
Conversely, HAART-related hepatotoxicity may enhance the progression of liver fibrosis.
      
Objective: To study the effect of Xuesaitong soft capsule (XST) on liver fibrosis criteria in patients with post-hepatitis fibrosis.Methods: Sixty-four patients with such fibrosis were randomly divided into the treated group and control group.
      
Their liver fibrosis criteria were examined before and after treatment.Results: The levels of serum procollagen III, hyaluronic acid, collagen IV, laminin in the two groups were significantly lower (P>amp;lt;0.
      
The differences between the two groups were insignificant (P>amp;gt;0.05).Conclusion: XST could recover liver dysfunction and had anti-liver fibrosis function.
      
However, response rates are much lower in patients who are difficult to treat, such as those infected with hepatitis C virus genotype 1, high viral load, or advanced liver fibrosis.
      
Can we identify liver fibrosis in HCV-infected patients without a liver biopsy
      
The most relevant consequence of persistent infection with the hepatitis C virus is liver fibrosis, which will evolve into cirrhosis in a significant proportion of patients.
      
However, there is an increasing interest to use noninvasive methods for the diagnosis of liver fibrosis.
      
There have been several approaches aimed at identifying liver fibrosis.
      
HIV-1 impacts the course of HCV infection by increasing the rate of HCV viral persistence, quantitative viral loads, and liver fibrosis progression rate.
      
There is a clinical need for noninvasive measurement of liver fibrosis both to diagnose significant liver fibrosis and to monitor the effects of therapy on fibrogenesis and fibrolysis.
      
However, response rates are disappointing in patients who are difficult to treat, such as those infected with HCV genotype 1, high viral load, or advanced liver fibrosis.
      
Whether metabolic steatosis has a direct role in liver fibrosis progression or is only a surrogate marker of an underlying defect triggering fibrogenesis, such as insulin resistance, is a subject of debate.
      
HIV-1 has deleterious effects on the course of HCV infection by increasing the rate of HCV viral persistence, quantitative HCV RNA levels, and ultimately the liver fibrosis progression rate.
      
If untreated, long-term infection may cause anemia or undernutrition and may progress to liver fibrosis, portal hypertension, or hydronephrosis.
      
Studies with strong rationales include those investigating high-dose peginterferon/ribavirin, long-term peginterferon suppression, potential immune modulators, and potential inhibitors of liver fibrosis.
      
Advanced liver fibrosis and portal hypertension due to chronic schistosomiasis are irreversible.
      
The pathogenesis of liver fibrosis in schistosomiasis japonica: Sequential qualitative and quantitative immunohistochemical stud
      
Study on the role of mast cells in the development of liver fibrosis during diethylnitrosamine (DEN) -induced hepatocarcinogenes
      
The mast cells (MC) and their roles in the development of liver fibrosis during experimental hepatocarcinogenesis in rats have been studied immunohistochemically and electron microscopically.
      
 

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