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ulcers
Beh?et's disease is a systemic vasculitis characterized by recurrent oral and genital ulcers and ocular inflammation.
      
Ocular manifestations include anterior uveitis, posterior uveitis, macular edema, retinal vasculitis, conjunctival ulcers, optic neuropathy, papilledema, secondary glaucoma, cataract, and neovascularization of iris or retina.
      
Many recent studies have evaluated thalidomide in patients with HIV infection, in which this drug is an efficacious agent against oral aphthous ulcers, HIV-associated wasting syndrome, HIV-related diarrhea, and Kaposi's sarcoma.
      
However, these drugs are associated with an increased risk for the development of adverse sequelae, including serious upper gastrointestinal side effects such as symptomatic ulcers, perforation, obstruction, and gastrointestinal bleeding.
      
Gastroduodenal ulcers detected by endoscopy are markedly diminished in patients receiving selective COX-2 inhibitors versus those receiving NSAIDs.
      
Moreover, unpublished data indicate that the risk of symptomatic and complicated ulcers is reduced by approximately half in patients prescribed rofecoxib or celecoxib.
      
pylori infection leads to a decreased risk for endoscopic ulcers in people taking NSAIDs.
      
New hope for systemic sclerosis patients with digital ulcers
      
Beh?et's syndrome (BS) is a multisystem disease of unknown etiology, characterized by chronic relapsing cardinal symptoms of orogenital ulcers, uveitis, and different skin lesions.
      
Beh?et's disease is a chronic relapsing systemic vasculitis of small and large vessels, of unknown etiology, characterized by recurrent oral and genital ulcers, and ocular inflammation.
      
Venous ulcers resulting from primary saphenous vein insufficiency, which account for 50% of all venous ulcers, and recurrent venous ulcers should all be a thing of the past, apart from those associated with deep valvular insufficiency disease.
      
Endovascular and surgical techniques aimed at treatment of primary and secondary venous valvular reflux have been shown to improve venous hemodynamics promoting healing of venous ulcers and improving quality of life.
      
They also reduce NSAID-related dyspepsia.Specific cyclooxygenase-2 (COX-2) inhibitors are associated with a markedly reduced rate of endoscopic ulcers.
      
Both drugs demonstrate a reduced incidence of endoscopic ulcers.
      
Lesions responsible for small bowel bleeding, on the other hand, include arterio-venous malformations, tumors, and ulcers.
      
The same option is available for small bowel ulcers that show stigmata of recent bleeding, eg, a non-bleeding visible vessel or adherent clot, or are actively bleeding at the time of enteroscopy.
      
These include abdominal pain/dyspepsia in the absence of ulcer disease; symptomatic and asymptomatic gastric and duodenal ulcers; and clinically significant upper GI events such as bleeding, gastric outlet obstruction, and perforation.
      
Treatment of small intestinal ulcers depends on the cause of the ulcers.
      
Ulcerations of the small intestine (apart from duodenal ulcers) are uncommon.
      
Treatment of isolated nonspecific ulcers include discontinuation of medications known to cause nonspecific ulcerations, balloon dilation of strictures, and segmental resection of involved segments.
      
 

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