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ulcers
A recent placebo-controlled trial of high-dosage parenteral omeprazole after endoscopic treatment of bleeding peptic ulcers demonstrated a substantial reduction in the risk of rebleeding.
      
Stress-related mucosal disease (SRMD) includes stress-related injury (superficial mucosal damage) and stress ulcers (focal deep mucosal damage).
      
Prophylaxis of stress ulcers may reduce major bleeding but, so far, has not been shown to improve survival.
      
Treatment of peptic ulcers with adherent clots is currently controversial.
      
Hemoclips may be preferable for very deep ulcers and large visible blood vessels if coaptive coagulation is anticipated to have a high risk of perforation or bleeding.
      
Coxib therapy has better GI tolerance than NSAIDs, but patients with peptic ulcers or dyspepsia during NSAID/coxib treatment need PPI co-therapy.
      
An initial treatment interval of 4 to 8 weeks should allow for interim healing of iatrogenic ulcers.
      
Patients are routinely given standard doses of available proton-pump inhibitors (PPIs) to facilitate healing of iatrogenic ulcers and to prevent secondary bleeding.
      
Patients with ulcers that demonstrate only low-risk endoscopic stigmata (clean base or flat pigmented spot) can be treated with an oral PPI at double the standard clinical dose.
      
Patients with ulcers that demonstrate high-risk endoscopic stigmata (spurting, oozing, or nonbleeding visible vessel) should receive high-dose intravenous PPI treatment following appropriate endoscopic hemostatic treatment.
      
Patients with minor stigmata or clean-based ulcers will not benefit from endoscopic therapy and should be triaged to less intensive care and be considered for early discharge.
      
Although the investigation of acid-peptic disorders helped to launch the modern understanding of abdominal surgery, the majority of operative interventions are now aimed at managing emergent problems that arise from ulcers.
      
If an NSAID must be used in a patient at risk, the lowest-risk NSAID should be used with, in many cases, cotherapy to reduce the risk for ulcers.
      
The acid suppression provided by traditional doses of histamine 2-receptor antagonists (H2RAs) does not prevent most NSAID-related gastric ulcers.
      
Despite a single study demonstrating that H2RAs at double the dose may be effective, studies comparing such high doses with misoprostol or proton pump inhibitors (PPIs) for preventing NSAID ulcers are not available.
      
PPIs are effective at single daily doses, do not demonstrate tachyphylaxis, and are superior to H2RAs and misoprostol in reducing ulcers and NSAID-associated dyspepsia.
      
CD usually causes ulcers in the ileocoecal area, but any part of the gut may be affected.
      
The purpose of the study is to identify a repair procedure for the ulcers or defect of the anterior lateral plantar region.
      
The results showed that corneal shield ulcers and superficial punctuate keratitis healed during the first week after surgery and did not recur.
      
Gastroduodenal ulcers: Causes, diagnosis, prevention and treatment
      
 

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