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primary vitreous
A 12-year-old girl had an orbital lymphangioma and ipsilateral persistent hyperplastic primary vitreous (recently termed persistent fetal vasculature).
      
Acute angle-closure glaucoma in a man with persistent hyperplastic primary vitreous
      
Acute angle-closure glaucoma in this patient may have been associated with persistent hyperplastic primary vitreous.
      
Persistent hyperplastic primary vitreous (PHPV), a developmental cause of leukocoria, is due to incomplete regression of the fetal blood supply to the eye.
      
Ocular lesions included infection, trauma, retinal detachment, retinoblastoma, Coat's disease, and persistent hyperplastic primary vitreous.
      
In the rabbit the primary vitreous develops from the 11th to 26th day of gestation.
      
Free mesenchymal cells migrate into the vitreous cavity together with the developing vessels of the primary vitreous.
      
Observation of consecutive stages suggests that the first cells of the secondary vitreous are derived from the primary vitreous and not from the retinal or the ciliary body.
      
Unsolved is the problem how the primary vitreous and the hyaloid vessels desappear without leaving a trace.
      
Quite frequently there are more or less peripheral connecting branches between the developing definitive retinal vessels and the vessels of the primary vitreous.
      
The findings support the recommendation of a primary vitreous repair in severely injured eyes.
      
Ultrastructural pathology of anterior persistent hyperplastic primary vitreous
      
Using transmission electron microscopy, the fine structure of anterior persistent hyperplastic primary vitreous (PHPV) removed from the eyes of four infants was studied.
      
We believe that the retinal and other manifestations of Norrie disease are the result of a primary abnormality of vascular proliferation, probably in relation to persistent hyperplastic primary vitreous after approximately 14 weeks' gestation.
      
Early morphogenesis of persistent hyperplastic tunica vasculosa lentis and primary vitreous (PHTVL/PHPV)
      
This study provides scanning electron microscopic observations on the early morphogenesis of persistent hyperplastic tunica vasculosa lentis and primary vitreous (PHTVL/PHPV) in canine fetuses at days 28 35 postcoitum (D28 and D35).
      
There was corneal opacity with anterior segment dygenesis in the left eye, and persistent pupillary membrane, cataract and persistent hyperplastic primary vitreous in the right eye.
      
She had marked mental and physical retardation, generalized hypertonia, microphthalmia with persistent hypoplastic primary vitreous, blepharochalasia, high nasal bridge, micrognathia, malformed ears with preauricular pits, and overlying fingers.
      
At birth the patient presented with right microphthalmia, right microcornea, and persistent hyperplastic primary vitreous of the right eye.
      
Anomalous vitreous adhesion over the posterior retina could be related to the malformative genesis of the syndrome, and the anomalous persistence of the Cloquet's canal or primary vitreous could be responsible for the traction.
      
 

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