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We conclude that nimesulide does not interact with these drugs in terms of antiinflammatory action and antagonizes their side effects on gastric tissue without reacting chemically.
      
The anticholinergic activity was incorporated into the intact esters to overcome the gastric toxicity of indomethacin, not only by blocking the acidic functionality but also by decreasing gastric secretions and motility.
      
All the derivatives were significantly less irritating to the gastric mucosa than the parent drug.
      
MSI/LOH and extron expression of the FHIT gene in gastric carcinoma
      
There was no correlation between LOH and MSI of the FHIT gene in GC and the histological characteristics of gastric carcinoma (Bormann's or Lauren's classification).
      
The findings in this study suggest that LOH and MSI of FHIT gene may induce aberrant extron expression, which might play a role in gastric carcinogenesis.
      
The left gastric artery and coronary vein were ligated in all the cases.
      
Effect of oxytocin on gastric ischemia-reperfusion injury in rats
      
The effect of peripherally administered oxytocin (OT) on gastric ischemia-reperfusion injury (GI-RI) and its possible mechanism were investigated.
      
These results indicated that the oxytocin could significantly protect gastric mucosal against injury induced by ischemia-reperfusion, and the oxytocin receptor was involved.
      
This effect of oxytocin may be mediated through the vagus and sympathetic nerve, and then lead to the reduction of gastric juice output and the depression of gastric acidity.
      
To investigate the biological behavior of GIST, we collected 83 cases of gastric and 62 cases of small intestinal GIST from the Department of Pathology of the Chinese PLA General Hospital.
      
We find that the average age of gastric GIST was 55.4 years.
      
Small intestinal GIST was more frequently associated with metastasis and tumor relapse than gastric GIST (χ2 = 6.131, P = 0.013).
      
The COX hazard proportional model revealed that advanced clinical stage (P = 0.001), large tumor size (P = 0.001), a high mitotic index (P = 0.002) and the high risk grade (P = 0.018) indicated a poorer prognosis in gastric GIST.
      
The frequency of c-kit mutation was 32% and 22.5% for gastric and small intestinal GIST, respectively.
      
In gastric GIST, the mutated c-kit was predominant in patients older than 50 years of age.
      
In conclusion, for gastric GIST, clinical stage, tumor size, mitotic index, and risk grade are the prognostic indicators.
      
Small intestinal GIST are more aggressive than gastric GIST.
      
Proteinases from Gastric Mucosa of European Sheatfish, Silirus Glanis L.
      
 

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