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lead acetate
Removal of the soluble humic substances through their precipitation by a basic lead acetate (15.9 g/L Pb(CH3COO)2 · 3H2O-4.7 g/L PbO) solution corrected their interference effectively.
      
Solutions under study contain (0.5-2) × 10-5M lead acetate, 0.05-0.2 M KCN, 0.1 M KOH, and 0.01 M KAu(CN)2.
      
In experiments on male white mice we initiated the induction of peroxidation of lipids by lead acetate, tetrachloromethane, sodium nitrite, or SOVOL (a mixture of polychlorinated biphenyls).
      
The effects of pectin from the eelgrass Zostera marina on toxic liver injury induced by enteral administration of lead acetate are examined in experiments on rats.
      
The sorption of pyrene by a filter paper modified with lead acetate is studied by luminescence methods.
      
It involves the addition of lead acetate solution to the extract prior to its passage through the bonded-phase cartridge.
      
The aim of this paper was to determine whether prolonged drinking of lead acetate-containing water by adult rats, which imitates environmental exposure to lead (Pb), affects some morphological and biochemical properties of rat brain microvessels.
      
Lead acetate·3H2O was administered either in the diet for 3 wk at 2000 ppm Pb or by intraperitoneal (ip) injection of 3-wkold chicks with 52 mg Pb/100 g body wt.
      
A factorial experiment was conducted with weanling rats fed a purified diet to determine the influence of dietary lead (0 or 100 ppm) as either lead acetate or lead carbonate on fluoride bioavailability (2 or 10 ppm as sodium fluoride).
      
Dams were administered Pb as lead acetate (PbAc), either through their drinking water (500 ppm PbAc) or through twice daily intubations (3 mg PbAc/Kg body wt) from postpartum d 1 (P1) to P21 (P0=day of birth).
      
In two fully crossed, two-factor experiments, F1 generation male rats were fed a basal diet supplemented with lead (lead acetate) at 0 or 2 μg/g and iron (ferric sulfate) at 50 or 250 μg/g (Experiment 1).
      
Effect on blood, liver, and kidney variables of age and of dosing rats with lead acetate orally or via the drinking water
      
Levels of lead in the livers and kidneys of rats increased in proportion to the dose of lead acetate that the rats were given orally or in the drinking water.
      
Effect of diet on the response in rats to lead acetate given orally or in the drinking water
      
Liver lead levels were higher for rats that were orally dosed with 100 mg lead acetate/kg body wt and fed a semipurified diet than those fed a pelleted diet.
      
The levels of glutathione in the liver decreased in response to lead acetate in the drinking water of rats fed the semipurified diet, but not in the livers from the group fed the pelleted diet and treated with lead.
      
The levels of lead in the kidneys were higher in the group given lead acetate in their drinking water and fed the semipurified diet than in the lead treated group fed the pelleted diet.
      
The objective was to examine the effects of dietary oil (cottonseed oil vs fish oil), dietary antioxidant (0 vs 75 ppm ethoxyquin), and dietary lead (0 vs 1000 ppm Pb as lead acetate trihydrate) on hepatic fatty acid composition.
      
Lead levels in bone and hair of rats treated with lead acetate
      
For 12-16 wk rats were given tap water containing lead acetate in the following concentrations: 41.7 mg Pb/L, 83.3 mg Pb/L, and 166.6 mg Pb/L.
      
 

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