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serum igg
The affinity constants of protein A binding to the CH2-CH3 domain interface demonstrated differences between serum IgG1 and tumor associated IgG1 that did not exceed 3-8-fold.
      
The binding affinity toward the anti-CH2 monoclonal antibody determined for serum IgG1 and IgG1 from carcinoma cells differed not more than 2.5-fold.
      
The level of serum IgG was supposed to be a main factor that affected the final protection of challenge.
      
20 μg recombinant protein could induce an endpoint titre of serum IgG as high as 51200, which was enough to afford 100% protection against 400 LD50 virulent 141 challenge.
      
Serum IgG antibody levels (by ELISA) as well as effects on daily weight gain were measured on 0, 7, 14 and 28th day after immunization.
      
Serum IgG, IgA and IgM concentration in 1,038 patients with various neurological disorders
      
Concentration of serum IgG, IgA and IgM in 1,038 unselected patients with various neurological diseases was determined.
      
Selective quantitative abnormalities in serum IgG were observed in 35% of patients with subacute sclerosing panencephalitis as compared to 5% in instances in the "definite" MS group.
      
Most frequent quantitative abnormalities in serum IgG, IgA and/or IgM were established in malignant lymphoproliferative disorders, in patients with myopathies including myositides and in subacute or chronic inflammatory CNS disorders.
      
Isoelectric focusing and quantitative estimation of cerebrospinal fluid and serum IgG in idiopathic polyneuropathy
      
Acute IP very frequently had an increase of oligoclonal and/or polyclonal serum IgG during the progressive phase and blood-CSF barrier damage accompanied by polyclonal IgG intrathecal synthesis during the stationary phase.
      
Isoelectric focusing of CSF and serum IgG followed by crossed immuno isoelectric focusing and direct immunofixation as well as quantitative assay of IgG and albumin were performed in 64 clinically definite multiple sclerosis patients.
      
The clinical effect and the effect on serum IgG antibodies to peripheral nerve tissue of plasma exchange in patients with Guilla
      
Association of HLA-DR2 antigen with serum IgG antibodies against Borrelia burgdorferi in Bannwarth's syndrome
      
At the follow-up studies, 33 patients showed no, and 23 only mild, clinical residual symptoms including normal CSF findings and low-positive serum IgG borrelia antibody titres (IFT; ELISA).
      
Polyclonal elevations of serum IgG and/or IgA were seen in 8 patients.
      
The following immunological parameters were investigated: peripheral blood lymphocyte subsets, natural killer activity, serum IgG, IgM, ICAM-1 and tumour necrosis factor alpha (TNF-α).
      
Serum and CSF IgG antibody responses to αB-crystallin were significantly elevated only in NBD patients (serum IgG, NBD 1.29±0.49 vs NIND 0.95±0.39, p=0.01; CSF IgG, NBD 1.22±0.64 vs.
      
every five weeks, a mean increase in the preinfusion serum IgG level of 0.3 g/l was observed, as compared to earlier i.m.
      
Finally, 100 mg/kg/week every three weeks gave 5/5 patients a preinfusion serum IgG level of >amp;gt;4 g/l with a mean rise of 3.6 g/l, as compared with the levels before the study.
      
 

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