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cardiac disease
Thyrotoxicosis is not only able to aggravate preexisting cardiac disease but can also by itself lead to atrial fibrillation, cardiomegaly or congestive heart failure.
      
Of major importance is the investigation of the underlying cardiac disease.
      
Heart transplantation is in our opinion a reconsiderable alternative in the treatment of complex cardiac disease and cardiomyopathy in infants.
      
The number of women with congenital cardiac disease, who mature into adulthood is increasing.
      
Insomnia in patients with heart transplantation and cardiac disease is a common problem.
      
With regard to cardiac disease, treatment with mirtazapine, nefazodone or trazodone should be preferred because of the chinidine-like effect of tricyclic antidepressants (TCA).
      
The risks and benefits of hypnotic drugs are discussed especially in relation to pharmacological interaction (cytochrome system) and cardiac disease.
      
Pregnancy in congenital cardiac disease: an increasing challenge for cardiologists and obstetricians - a prospective multicente
      
Method: We measured the bidirectional P wave signal averaged ECG of 45 consecutive patients with (group A) and without (group B) paroxysmal atrial fibrillation (PAF) and 15 young volunteers without a cardiac disease (group C).
      
A 53-year old woman without a previous history of cardiac disease was successfully resuscitated from ventricular fibrillation.
      
Both clinically and morphologically, the cardiac disease was the cause of death in this patient with FSHD.
      
In all stages of cardiac disease, the primary goal is to reach near-normoglycemia.
      
Elevated concentrations of B-type natriuretic peptide (BNP) and N-terminal pro- BNP (NT-proBNP) reflect elevated myocardial wall stress due to volume or pressure overload in cardiac disease.
      
In ischemic cardiac disease, hereditary cardiomyopathies and myocarditis, the hypertrophy compensates for the functional insufficiency of the damaged myocardial tissue and is designated as hypertrophy due to damage.
      
Knowledge concerning the regulation of Na,K-ATPase and its link to K handling may render new insight in the pathophysiology of cardiac disease, and may hence lead to novel therapeutic interventions based on a rational approach.
      
The precise correlation between magnesium and cardiac disease remains to be established.
      
Matrix metalloproteinases (MMPs) are important in cardiac disease such as heart failure.
      
About every sixth patient dies of sudden cardiac death in the period after pacemaker implantation, whereby death is in the vast majority of cases ascribable to progression of underlying cardiac disease.
      
Therapy with permanent pacing devices affects neurohumoral reactions with regard to the underlying bradycardia mechanism, cardiac disease and pacing mode.
      
Impairment of muscle function was mild and slowly progressive, whereas the cardiac disease was severe and rapidly progressive.
      
 

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