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prostate cancer
The possibility of using AGR2 as a diagnostic marker of prostate cancer is discussed.
      
Calcitriol induces transcription of the placental transforming growth factor β gene in prostate cancer cells via an androgen-ind
      
Calcitriol (1α,25-dihydroxycholecalciferol) suppresses the growth of prostate cancer cells.
      
Growth suppression of hormone-sensitive LNCaP prostate cancer cells by calcitriol is believed to depend on androgens, but the mechanisms of the interactions between the calcitriol-and androgen-dependent signaling pathways is unclear.
      
It was assumed that calcitriol stimulates production of PTGF-β independently of 5α-dihydrotestosterone and that its effect on prostate cancer cell growth is partly mediated by an androgen-independent mechanism.
      
Aberrant methylation of p16, HIC1, N33, and GSTP1 in tumor epithelium and tumor-associated cells in prostate cancer
      
Loss of heterozygosity and microsatellite instability in tumor-associated stromal cells and tumor epithelium of prostate cancer
      
The epithelial and stromal components of prostate cancer (PC) and prostatic intraepithelial neoplasia (PIN) were isolated by laser capture microdissection and subjected to microsatellite analysis of chromosome regions 8p22, 13q14, and 16q23.
      
The goal of this study was to optimize the synthesis of [N-methyl-11C]choline, a radiopharmaceutical used in the diagnosis of brain tumors and prostate cancer with positron emission tomography (PET).
      
The review highlights analytic capacities of immunochemical and molecular-genetic methods used in the non-invasive test for prostate cancer and monitoring of efficacy of anticancer therapy.
      
It was proved that there was a putative predisposing gene for prostate cancer in this region, and that analogs of GGPP can inhibit the geranylgeranylation of p21rap protein in PC-3 prostate cancer cell lines.
      
These facts suggest thatGGPPS may be one of the candidate genes for prostate cancer.
      
The anti-proliferative effects of a branched chain fatty acid from a sea cucumber on prostate cancer cells was reported with the activity of 5-lipoxygenase.
      
Following incubation, the cultured cells containing immature DCs were concentrated and transfected with tumour mRNA from human prostate cancer cell lines employing a highly efficient electroporation procedure.
      
Thawed transfected DCs were able to elicit primary T-cell responses in vitro against antigens encoded by the prostate cancer mRNA as shown by ELISPOT assay using mock-transfected DCs as control.
      
Deregulation of AR signalling may be a causal factor in prostate cancer development.
      
AR is the main target in prostate cancer therapies.
      
Genistein inhibits the contact-stimulated migration of prostate cancer cells
      
The results of several epidemiological studies have suggested that a soybean-based diet is associated with a lower risk of prostate cancer.
      
However, the central finding of this study is that at physiologically relevant concentrations (1 μM and 10 μM), genistein inhibits the motility of prostate cancer cells stimulated by homo-and heterotypic contacts.
      
 

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