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chronic type b
The second patient (male, 71years) had a chronic type B aortic dissection and developed dynamic compression of true lumen and thus peripheral malperfusion.
      
Alternatively, the discrepancy of response might relate to the natural course of chronic type B hepatitis.
      
Accordingly, controlled trial is mandatory for assessing the effect of Ara-A or any other agent in the treatment of chronic type B hepatitis.
      
Acute hepatitis C virus superinfection followed by spontaneous hepatitis B e antigen seroconversion and hepatitis B surface antigen clearance in a patient with chronic type B hepatitis is described.
      
Twenty-two patients with chronic type B hepatitis were treated with OK-432.
      
These data suggest that HBsAg is at least one of the target antigens expressed on the hepatocyte membrane possibly enabling cytolytic interaction by cytotoxic T cells in chronic type B hepatitis.
      
IgM antibody to hepatitis B core antigen in children with chronic type B hepatitis
      
Chronic type B hepatitis and primary hepatocellular carcinoma in children
      
Fifty-three had acute type A(Ac-A), 29 had chronic type A(Ch-A), 20 had acute type B(Ac-B), and 23 had chronic type B(Ch-B) dissections.
      
Treatment of Chronic Type B Aortic Dissection with Endovascular Stent-Graft Placement
      
The indication for thoracic aortic repair was atherosclerotic ulcer in 7 cases, chronic aortic aneurysm in 18, acute type B dissection in 5, and chronic type B dissection in 12.
      
Endovascular Fenestration for Relief of Chronic Mesenteric Ischemia from Chronic Type B Aortic Dissection
      
Serum 2'-5'oligoadenylate synthetase as a monitoring marker of antiviral effect during interferon therapy for chronic type B hep
      
We evaluated the usefulness of serum 2'-5'oligoadenylate synthetase (2-5AS) activity assay in monitoring the anti-viral activity of chronic type B hepatitis patients during IFN therapy.
      
The results suggest that the serum 2-5AS activity assay is a useful probe for monitoring the anti-viral activity of chronic type B hepatitis patients during interferon therapy.
      
Interferon α and γ positive mononuclear cells and HLA antigen expression on hepatocyte membrane in the liver in chronic type B a
      
TA-NB-2 cells significantly lysed hepatocytes from 22 of 23 patients with chronic NANB hepatitis, whereas they lysed hepatocytes from only one of 17 control patients with chronic type B hepatitis, acute hepatitis B or acute hepatitis A.
      
Spontaneous hepatitis B surface antigen clearance in a long-term follow-up study of patients with chronic type B hepatitis.
      
A chronic type B dissection revealed impending rupture due to false lumen infection 1 year after operation and repeat surgery was performed.
      
A viral mechanism in acute exacerbations of chronic type B hepatitis: Hepatitis B virus reinfection and subsequent reactivation
      
 

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