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chronic heart failure
Twenty-seven CHD patients had coronary insufficiency but not heart failure and had a left ventricle ejection fraction of ≥50%; 26 patients had chronic heart failure and an ejection fraction of >amp;lt;40%.
      
Association of polymorphic markers I/D of gene ACE and A1166C of gene AT2R1 with ischemic chronic heart failure in the Russian a
      
Alterations of cardiac and lymphocyte β- adrenoceptors in rat with chronic heart failure
      
The alterations of cardiac and lymphocyte β-adrenoceptors were observed in the rats with chronic heart failure produced by constriction of both abdominal aorta and renal artery.
      
chronic heart failure, left ventricular dysfunction and coronary artery disease.
      
Acute heart failure consists of decompensated chronic heart failure, acute pulmonary edema and cardiogenic shock.
      
The ventilatory and cardiac chemoreflex sensitivities are tools which could improve prediction of malignant cardiac arrhythmias and the prognosis of patients with advanced chronic heart failure.
      
Despite medical therapy, chronic heart failure can often only be treated using a mechanical assist system.
      
We assessed the influence of the ACE-inhibitor Quinaprilat and the vasodilator sodium nitroprusside (NPN) on the catecholamine plasma levels in 22 patients with chronic heart failure.
      
Methods Twenty-two patients with chronic heart failure (NYHA class II-IV) with a cardiac index >amp;lt;2.5 l/min×m2 and/or a pulmonary capillary wedge pressure >amp;gt;16 mmHg were randomized to receive Quinaprilat or NPN intravenously.
      
Conclusion In patients with chronic heart failure, intravenous treatment with Quinaprilat or NPN over 4 hours does not induce an acute decrease in plasma catecholamine levels.
      
Major goals in the treatment of chronic heart failure however include symptom alleviation, prevention of manifestation and progression of heart failure and thereby improving prognosis.
      
Chronic heart failure and cardiac cachexia and links between the endocrine and immune systems
      
Chronic heart failure (CCHF) is a complex syndrome affecting many body systems.
      
The main symptoms of chronic heart failure are dyspnoe and exercise intolerance.
      
Mortality of chronic heart failure in industrial countries remains unacceptably high despite advances in medical therapy.
      
Objective Advanced chronic heart failure is a hypercatabolic state with an imbalance between anabolic and catabolic metabolism and finally progressive loss of both muscle mass and adipose tissue.
      
Therfore, we tested the hypothesis that plasma leptin concentrations are reduced in advanced chronic heart failure.
      
Can serum NT-proBNP detect changes of functional capacity in patients with chronic heart failure
      
Beta blockers and glucose metabolism in chronic heart failure: Friend or foe
      
 

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