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by macrophages
Human plasma low-density- (LDL) and high-density lipoproteins (HDL) are still more potent inducers of cystatin C secretion by macrophages.
      
Phagocytosis of apoptotic bodies by macrophages was inhibited with sialooligosaccharide ligands of siglec-5 and MAbs to siglec-5.
      
It is shown that the NODMs under the action of nitrogen oxide metabolites generated by macrophages and similar cells in inflammations or infections should lead to a sharp increase in the number of mutations in the case of RNA-containing viruses.
      
coli cells ingested by macrophages increased rapidly for the initial 60 min of incubation at 37°C.
      
The uptake of nonopsonized bacteria by macrophages was significantly lower than that of the opsonized ones (p >amp;lt; 0.05).
      
It is found that the signal molecules of the down-stream of Ras, Raf-1, MAPK p44, and MAPK p42 are phosphorylated, and cPLA2 is activated with a significant increase of the release of [ H3 ] AA by macrophages in response to LPS and PMA.
      
Since 1αOhase can locally convert 25-hydroxyvitamin D into 1α,25-dihydroxyvitamin D (1,25(OH)2D), an active metabolite of vitamin D, it is suggested that local production of 1,25(OH)2D by macrophages may promote atherosclerotic calcification.
      
In some this may be mediated predominantly by lymphocytes; in others, where the demyelination is produced primarily by macrophages, the process may be antibody-mediated.
      
Efficient removal of myelin debris by macrophages may thus facilitate differentiation and permit successful remyelination of damaged axons.
      
When injected by the intravenous route, liposomes are taken up by macrophages in the liver and in the spleen.
      
Uptake of each drug by macrophages was markedly enhanced by liposomal encapsulation.
      
Defective reactive oxygen metabolite generation by macrophages from acute brucellosis patients
      
The production of nitric oxide (NO) by macrophages is important for the killing of intracellular pathogens, such asToxoplasma gondii.
      
In the early stage, FN might be produced by macrophages and sinusoidal cells, acting as a chemotactant for fibroblasts.
      
It has been demonstrated that insulin could augment the production of IL-1 by macrophages stimulated by LPS.
      
Both the uptake and degradation of125I-VLDL by macrophages were saturable, and the uptake and degradation curves were virtually identical.
      
In contrast, the binding, uptake and degradation of125I-β-VLDL by macrophages from diabetic mice were reduced to only about 45 % of normal levels because of a decrease in the number and affinity of the receptors for β-VLDL.
      
The augmentation of T cell activation by DATS was related to its inhibitory effect on the production of nitric oxide (NO) by macrophages.
      
In a wide range of concentrations (1-100 μg/ml), DATS can inhibit the production of NO by macrophages (P>amp;lt;0.05,P>amp;lt;0.01).
      
DATS, despite its inhibition of the production of NO by macrophages, can significantly enhance the production of hydrogen peroxide (H2O2) by macrophages.
      
 

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