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kidneys
If untreated and uncorrected, the result, with progressive frequency over a period of 10-15 years, is increased morbidity, especially with pregnancies, structural damage to the kidneys, kidney stones, uremia, hypertension, and premature death.
      
All patients were monitored for toxicity in the inner ear, kidneys, bone marrow, and liver.
      
The concentration of thiamphenicol in severely diseased human kidneys
      
The concentration of thiamphenicol in serum and renal tissue was determined in 17 patients with severely diseased kidneys after an intravenous injection of 1000 mg of the drug.
      
The highest renal tissue/serum concentration ratios of thiamphenicol were observed in patients with hydronephrotic kidneys and renal tumours, the lowest in cases of pyonephrosis.
      
The high renal tissue levels of thiamphenicol in patients with severely diseased kidneys fulfill an important condition for the antibacterial chemotherapy of kidney infections.
      
The effect of aminoglycosides on proximal tubular membranes of human kidneys
      
A technique is described to obtain renal interstitial fluid (RIF) from rabbits after implantation of diffusion chambers with permeable membranes of 0.45 μ porosity in both kidneys.
      
Our results show that this diffusion chamber technique can be useful in the pharmacokinetic examination of drugs, also with respect to their distribution in the kidneys.
      
Composition of fluids from diffusion chambers implanted in the soft tissue and kidneys of rabbits
      
The fluids from diffusion chambers implanted in soft tissue and kidneys of rabbits were analysed for total protein, albumin, enzymes, ions, glucose, creatinine, urea, uric acid, bilirubin and cholesterol.
      
The nephrotoxicity of cefotiam was determined on the basis of the number of tubular epithelial cells excreted, the malate dehydrogenase activity in the urine and the histological examination of the kidneys.
      
The histological examination of the kidneys at the end of both the dosing (5 days) and the recovery (7 days) periods revealed no pathological changes indicating nephrotoxicity.
      
The treatment was efficacious in all patients with infections which were negative in the antibody-coated bacteria test and not complicated by anatomic and/or functional abnormalities of the kidneys and urinary tract.
      
The factors responsible for the deficient bacterial clearence from the kidneys of these patients, and the genetic control, have not been identified.
      
In contrast, T and B lymphocyte and complement (C5) defects had little effect on the clearance ofEscherichia coli from the kidneys.
      
The effect of IgG was due to a marked enhancement ofin vivo phagocytosis, as demonstrated by monitoring bacterial numbers in the liver, spleen, lungs and kidneys.
      
The factors responsible for the deficient bacterial clearance from the kidneys of these patients, and the genetic control, have not been identified.
      
In contrast, T and B lymphocyte and complement (C5) defects had little effect on the clearance ofEscherichia coli from the kidneys.
      
Cephalexin and trimethoprim are absorbed at a slower rate; epicillin, azlocillin, ticarcillin sulfonamides were eliminated at a faster rate by the kidneys which was unique to patients with cystic fibrosis.
      
 

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