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kidneys
The compound is mainly excreted through the kidneys.
      
Residual tissue infection in surviving animals was observed with all agents, but was heaviest with fluconazole, especially in the kidneys.
      
CDZ was predominantly eliminated by the kidneys (80% of dose), a further 20% being excreted in the bile.
      
An autopsy revealed the presence of the microorganism in many organs including liver, kidneys, pancreas, spleen, lung, and brain.
      
After being immunized with E7C91G-HSP70 vaccine, there were no pathological changes found in livers, kidneys and spleens of the mice, which proves that the vaccine is quite safe.
      
The reason for their inefficacy in the elderly may be related to their inherent unfavorable effect on systemic hemodynamics and pathophysiologic findings in the arterial tree, heart, kidneys, brain, and on the metabolism of lipids and carbohydrates.
      
The results of several trials with angiotensin-converting enzyme (ACE) inhibitors have proven them to be the standard of care for diabetics and their kidneys.
      
This includes Na+ resorption at the expense of K+ excretion in such tissues as kidneys, colon, sweat, and salivary glands.
      
Examination of NE spillover in obesity has shown regional overactivity in the kidneys.
      
Activation of the renin-angiotensin and sympathetic nervous systems and physical compression of the kidneys appear to contribute to obesity-induced increases in sodium reabsorption and hypertension.
      
Microangiopathy affects the kidneys, eyes, and nerves.
      
Mineralocortiocoids act directly through their receptors in specific centers in the central nervous system, kidneys, heart, and vascular smooth muscle to mediate hemodynamic homeostasis.
      
Leptin also produces sympathoactivation to kidneys, hindlimb, and adrenal glands, indicating that the obesity-associated increase in sympathetic nerve activity could be due in part to these sympathetic effects of leptin.
      
A better understanding of aldosterone's actions in nonepithelial tissues should pave the way to better protection of organs at risk such as the kidneys, heart, and brain.
      
Special considerations for the obese patient, in addition to adequately controlling the blood pressure, include correction of the metabolic abnormalities and protection of the kidneys from further injury.
      
Nephron number in normal human kidneys has been shown to vary up to eightfold.
      
The kidneys play a central role in the long-term regulation of blood pressure and in the pathogenesis of hypertension.
      
It was hypothesized that such activation occurs in the kidneys of hypertensive rats and causes tissue renin-angiotensin system (RAS)activation and end-organ damage.
      
New aspects of the relationship among hypertension, obesity, and the kidneys
      
An expanded intravascular (plasma) volume is associated with obesity, which is related to an increased venous return to the heart, increased cardiac output, and increased blood flow to kidneys and other organs in normal and hypertensive patients.
      
 

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