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globoid cell
Genetic galactosylceramidase deficiency (Globoid cell leukodystrophy, Krabbe disease) in different mammalian species
      
Globoid cell leukodystrophy (Krabbe disease) in man is a rare genetic disorder caused by deficiency of galactosylceramidase activity.
      
Thus, a deficiency of galactocerebroside β-galactosidase results in globoid cell leukodystrophy, whereas a reduced activity of arylsulfatase A is responsible for metachromatic leukodystrophy.
      
The MR findings in an adult patient with globoid cell leucodystrophy (GLD) or Krabbe's disease are presented.
      
Late-onset globoid cell leukodystrophy mimicking an infiltrating glioma
      
Subsequent brain biopsy established the correct diagnosis of globoid cell leukodystrophy (GCL), which suggests that the radiographic appearance of late-onset GCL may mimic that of an infiltrating glioma.
      
Neuropathological studies of two young dogs with globoid cell leucodystrophy revealed changes similar to those reported in human cases of this condition.
      
Peripheral neuropathy in globoid cell leucodystrophy (Morbus Krabbe)
      
Biopsy and autopsy material, from the peripheral nervous system of six subjects with globoid cell leucodystrophy (Krabbe's disease) was subjected to detailed light microscopic examination.
      
Sub-plasmalemmal linear densities of variable length (0.1≈1.0 μ) were found to be a constant feature of globoid cells in human as well as in canine globoid cell leukodystrophy (GLD).
      
Twins with high-risk globoid cell leukodystrophy (GLD) at the 6th gestational month were studied morphologically and enzymatically.
      
Globoid cell leukodystrophy: Specialized contact of globoid cell with astrocyte in the brain of twitcher mouse
      
1977 a) were the membrane specializations observed in globoid cells in globoid cell leukodystrophy (GLD) and in the cells of the mononuclear phagocytic system (Kawanami et al.
      
Morphological alterations of oligodendroglia were investigated in the spinal cord of the twitcher mouse, an authentic murine model of human globoid cell leukodystrophy (GLD) from day 5 to day 45 postnatal (p.n.).
      
A case of canine globoid cell leukodystrophy (GLD) was also included under the assumption that this disease was the same in the dog as in man.
      
Demyelination in the spinal cord of murine globoid cell leukodystrophy (the twitcher mouse)
      
Chronologic events of demyelination were investigated in the spinal cord of the twicher mouse, an authentic murine model of human globoid cell leukodystrophy (GLD) from 5 to 45 days postnatal.
      
Characteristic inclusions in the kidney of canine globoid cell leukodystrophy
      
The kidney of a 7-month-old male Cairn terrier with globoid cell leukodystrophy (GLD) was investigated with light and electron microscopes.
      
Lectin-histochemical studies were performed on paraffin-embedded brain tissue sections to identify the specific sugar residues of undegraded "stored" substances in the cytoplasm of globoid cells from patients with globoid cell leukodystrophy.
      
 

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