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The muscle tissue of mdx mice has genetic defects causing chronic dystrophic processes in it.
      
The muscle tissue of young mdx mice proved to retain a relatively high capacity for regeneration.
      
Under conditions of tissue therapy of the wound, the formation of muscle fibers from muscle cells of the graft and active regeneration of muscle fibers in the recipient mice were observed, and no structural defects were detected in the thymus.
      
The capacity of posttraumatic regeneration in old mdx mice was lower.
      
Antioxidant Systems in Tissues of Senescence Accelerated Mice
      
The LPS showed low toxicity in D-galactosamine-sensitized mice and low biological activity in plants.
      
Imbalance in the Enzymatic System of Production and Consumption of Active Oxygen Species in Liver of AKR Mice with Spontaneous L
      
It was found that in the latent period of leucosis (mice of 3-6 months of age) the glutathione reductase (GR) activity in cytosol and mitochondria decreased and v in SMP increased.
      
In the stage of developed leucosis (mice of 7-9 months of age), non-synchronous changes in the antioxidant system resulting in the shift of metabolism towards the prooxidant state were found.
      
Peritoneal evoked neutrophils of NMRI strain mice were incubated with 0.01-100 nM insulin for 1-60 min at 22, 30, or 37°C and activated by 0.1-50 μM N-formyl-methionyl-leucyl-phenylalanine (fMLP).
      
Neutral Mg2+-dependent sphingomyelinase (SMase) activity and LPO level were monitored in heart, kidney, and liver of mice after administration of BR.
      
BR injected to mice causes simultaneous and unidirectional alterations in both LPO level and SMase activity with a significant (p >amp;lt; 0.05) positive linear correlation between these two parameters.
      
Gene Targeting of Tissue Factor, Factor X, and Factor VII in Mice: Their Involvement in Embryonic Development
      
Nearly all genes involved in the blood coagulation system have been knocked out in mice.
      
Factor X (FX) deficiency causes partial embryonic lethality between E11.5-12.5.FX-/- mice that were born died from fatal neonatal bleeding.
      
Furthermore, the temporal and topographic expression pattern of MMP components, as well as studies in gene-deficient mice, suggest a functional role in neointima formation after vascular injury.
      
The different approaches to induction of catalytic response toward recombinant gp120 HIV-1 surface protein in mice with various autoimmune pathologies are described.
      
The peptidylphosphonate conjugate containing structural part of gp120 molecule is used for reactive immunization of NZB/NZW F1, MRL, and SJL mice.
      
Increased proteolytic activity of polyclonal antibodies in SJL mice encouraged us to investigate the production of antigen-specific catalytic antibodies on the background of induced experimental autoimmune encephalomyelitis (EAE).
      
The immunization of autoimmune-prone mice with the engineered fusions containing the fragments of gp120 and encephalitogenic epitope of myelin basic protein (MBP89-104) was made.
      
 

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