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retinoic acid treatment
In chondrocytes dedifferentiated by subculture or retinoic acid treatment, this transglutaminase appeared to be downregulated, while type II transglutaminase expression was induced.
      
Pretreatment of cells with staurosporine, a protein kinase C (PKC) inhibitor, was found to potentiate the granulocytic differentiation induced by a brief (2 h) retinoic acid treatment.
      
Transiently increased autophosphorylation of EGF-R after 48 h of retinoic acid treatment correlated with enhancement of EGF binding capacity on the H460 cell surface.
      
A newborn lethal defect due to inactivation of retinaldehyde dehydrogenase type 3 is prevented by maternal retinoic acid treatment.
      
A third patient, also with large pleural effusions and hypoxia, died 1 day after discontinuing 9-cis-retinoic acid treatment.
      
A similar concern was whether differentiation induced by retinoic acid treatment with or without cyclic AMP affected the recorded sodium currents.
      
All-trans-retinoic acid treatment of acute promyelocytic leukemia cells in vitro.
      
At the conclusion of 5 months of 13-cis-retinoic acid treatment, this patient continued to have multiple sites of disease detectable only by MIBG.
      
Full osteoblastic differentiation resulted from combining retinoic acid treatment with high level BMP signaling.
      
Genetics of APL and the molecular basis of retinoic acid treatment.
      
Here, we found that retinoic acid treatment reduces serum-stimulated c-fos expression.
      
InWestern blot analysis, the expression of other nonspecific proteins in cells was not influenced by retinoic acid treatment.
      
In particular, retinoic acid treatment essentially abolishes Xotx2 expression in neuroectoderm.
      
Moreover, it provides a mechanism for creating tiation by retinoic acid treatment.
      
More dramatic morphological abnormalities were observed with 10-4 M retinoic acid treatment, often resulting in the absence of a heart tube.
      
NT2 cells can differentiate into postmitotic neurons after retinoic acid treatment.
      
Retinoic acid treatment partially rescues failed septation in rats and mice.
      
Retinoic acid treatment alters the distribution of retinoic acid receptor-B transcripts in the embryonic chick face.
      
Retinoic acid treatment also inhibits F9 cell growth, but to a smaller degree than PD98059.
      
Retinoic acid treatment does not always result inTM expression in allcell types as was demonstrated in K562 and HSD cells.
      
 

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