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Studies in rats revealed dose dependency/ non-linearity in arteether pharmacokinetics with in the dose levels used.
      
In this study, we find that prodigiosin could effectively inhibit the proliferation of human pancreatic cancer cells H8898 in a dose-and-time-dependent manner, with an IC50 of 75μmol according to the results of MTT and cell proliferation assays.
      
Prodigiosin may effectively enter cells and promote the level of intracellular reactive oxygen species (ROSin) in a dose-dependent manner.
      
Doses ranging from 12.5-50 μM provided a dose-dependent reduction in the number of viable cells when compared to controls.
      
At the 50 μM dose, the number of viable cells was reduced by 65% suggesting potential antineoplastic effects of 9-O-methylfusarubin.
      
32% or less, have been evaluated in a full panel of 60 human cancer cell lines over a 5-log dose range at the National Cancer Institute.
      
While ranitidine at 100 mg kg-1 dose prevented diclofenac-induced ulcer formation, it reduced meloxicam-induced ulcer formation significantly.
      
Though only two esters showed antiinflammatory activity similar to that of the parent drug at equivalent dose levels, all the esters were equipotent to indomethacin in the mouse acetic acid-induced writhing assay for analgesic action.
      
It was found that the product with higher graft degree (Gd)(0.19%) and relatively excellent mechanical properties can be produced if the mass ratio of PP/rPP/AA is 90:10:0.8, where the selected pre-irradiation dose of rPP is 4 kGy.
      
The antifeedant rate in choice test reached 62%-86% at the dose 2.5 mg/mL, while in non-choice bioassay the rate was only 20%-29%.
      
In the second day after treatment with the dose at 10 mg/mL of the alkaloid, the RGR reduced by 39.8%, and the food intake and the feces weight were respectively 57.7% and 57.4% of the controls.
      
Sublethally-irradiated NOD/SCID mice were transplanted with ex vivo expanded HSPCs with the dose of 8.5 × 106 cells per mouse.
      
Surprisingly, FCM also determined that the percentage of hMSCs in G2/M-and S-phases also increased in a dose-dependent manner with respect to MDA concentration.
      
The mechanism of low-dose long-term exposed to SO2 is more complex.
      
The best dose of UV-B radiation is about 1.163 kJ·m-2.
      
When the dose of UV-B radiation is more than it, the effects of UV-B will be reduced.
      
According to the percentage of damaged DNA with tail and its TL/D (tail length to diameter of nucleus) value, the relationship between DNA damage degree and heavy metal dose and exposure time was determined.
      
Results showed that compared with control groups, UVB was able to induce HIF1α and TfR protein expression in a dose-and time-dependent manner in HaCat cells (P >amp;lt; 0.05).
      
TfR mRNA was expressed in a dose-dependent manner and reached a peak at the 8th hour in HaCat cells (P >amp;lt; 0.05) whereas HIF-1α mRNA expression was not affected by UVB treatment (P>amp;gt;0.05).
      
PD153035, a selective inhibitor of EGFR tyrosine kinase, inhibited the TfR protein expression in UVB-treated cells in a dose-dependent manner (P >amp;lt; 0.05).
      
 

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