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liver cell
A hypothetical mechanism of adaptation of rat liver cell metabolism to a deficiency of glucose as the main energy substrate is proposed.
      
After repeated injections of DNA-poly(L-Lys)Gal complexes, the level of human ApoA-I in rat serum increases, probably, due to accumulation of functional human apoA-I gene in the liver cell nuclei.
      
The OAT-dependent suppression proved to be associated with a decrease in the DNA-binding activity of HNF3 in liver cell extracts.
      
Expression of different liver cell markers during the early prenatal human development
      
The expression of the liver cell markers, vimentin, desmin, cytokeratins 7, 18, 19, and stem cell markers CD34 and Bcl-2 in the early stages of the human prenatal development was studied.
      
Such structures of BHK-21 cell and mouse liver cell are similar to that of HeLa cell.
      
Oligonucleotide of cFIX cDNA (canine FIX, cFIX) was used to transcript mRNA of dog liver cell to cDNA by RT-PCR, and further construct it on the plasmid vector pGEM-T.
      
Gene expression profile in immunologically injured liver cell of mice
      
We have shown the fetal liver cell engraftments into multiple tissues of adult healthy mice, achieved without suppressing the animals' immune systems.
      
The different noxious agents lead to hepatocellular necrosis and/or apoptosis with loss of liver cell specific functions subsequent to a fall of functioning hepatocytes below a critical number.
      
Progress has been made in the temporary substitution of specific liver cell functions bridging the time period between liver failure and resumption of hepatocellular functions or availability of a donor liver.
      
Alpha-fetoprotein and liver cell proliferation in rats fed choline-deficient diet
      
A significant increase in the rate of liver cell proliferation was evidenced in animals on the deficient diet.
      
It is suggested that increased synthesis of AFP corresponds to liver cell regeneration secondary to hepatic damage.
      
Quantitative studies on the DNA content and morphometric features of liver cell dysplasia
      
Differential proteomics in malignant and normal liver cell lines
      
Hepatitis C virus causes liver cancer by establishing chronic inflammation and proliferation of liver cells and by interacting with cell genes involved in liver cell regulation.
      
Normally, hepatic stores of glutathione combine with the toxic metabolite and prevent liver cell injury.
      
When glutathione stores are depleted by overproduction of this metabolite, however, the reactive metabolite binds to liver cell proteins and causes hepatic necrosis.
      
Nonalcoholic steatohepatitis (NASH) is one entity in a spectrum of chronic liver disease related to obesity, hyperinsulinemia, insulin resistance, and liver cell injury from free fatty acid toxicity or other oxidant stress.
      
 

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