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gi protein
These reductions for the depressed patients in Gs and Gi protein function were similar for unipolar and bipolar depression.
      
These results suggest that the inhibition of the Gi protein might be the key feature that controls signal inhibition.
      
These results suggest that CNP acts via the Gi protein and adenylyl cyclase, rather than through cGMP-dependent phosphodiesterase.
      
The calcium flux returned to the basal level within 60 s, a typical characteristic of Gi protein-mediated transient response.
      
We conclude that the protective effect of 2-ARs is mediated through coupling to the Gi protein.
      
We showed that this SR 144528 inhibitory effect on Gi-dependent receptors was mediated by a direct Gi protein inhibition through CB2 receptors.
      
 

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