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cardiac fibrosis
We found that noise stress significantly increased the microvessel wall area, the number of microvessels with an outer diameter >amp;gt;19 μm, the degree of cardiac fibrosis, and the extent of ischemic myocardial lesions in SHR, but not in NWR.
      
TNFα-antagonism attenuates the development of experimental diabetic cardiomyopathy associated with a reduction of intramyocardial inflammation and cardiac fibrosis.
      
Cardiac Fibrosis as a Cause of Diastolic Dysfunction
      
The interaction of coronary tone and cardiac fibrosis
      
Aldosterone has direct effects on the vasculature and has been associated with vascular smooth muscle cell hypertrophy, endothelial dysfunction, cardiac fibrosis, proteinuria, and renal vascular injury.
      
U-II induces cardiac fibrosis by increasing fibroblast collagen synthesis.
      
We have shown that U-II antagonism using a selective U-II blocker, SB-611812, attenuates cardiac dysfunction by decreasing cardiomyocyte hypertrophy and cardiac fibrosis.
      
Pattern of cardiac fibrosis in rabbits periodically fed a magnesium-restricted diet and administered rare earth chloride through
      
It is suggested that the element may trigger a wound-healing response in the cardiac tissue leading to cardiac fibrosis.
      
In vivo, iron overload is associated with cardiac fibrosis.
      
AZT alone caused mild cardiac lesions, resulting in partial cardiac fibrosis, especially in the atrium.
      
In conclusion, stimulation of the overexpressed human AT2R in porcine cardiac fibroblasts inhibited tyrosine phosphatase activity but had no significant effect on fibroblast functions related to cardiac fibrosis.
      
Angiotensin (Ang) II is a key player in left ventricular (LV) remodeling and cardiac fibrosis.
      
From several studies, it is evident that AngII enhances TGF-β1 expression via activator protein 1 (AP-1) activation, and that this pathway is involved in hypertrophic growth of the heart muscle and in the development of cardiac fibrosis.
      
Cardiac effects of mineralocorticoids include stimulation of matrix protein deposition leading to cardiac fibrosis, which is at least partially due to the direct action of the hormones on cardiac cells.
      
The signaling mechanisms mediating mineralocorticoid-induced cardiac fibrosis have so far remained elusive.
      
This DOCA/high-salt treatment increased blood pressure in both genotypes but led to significant cardiac fibrosis only in sgk1+/+ but not in sgk1-/- mice.
      
Our results suggest that SGK1 plays a decisive role in mineralocorticoid-induced CTGF expression and cardiac fibrosis.
      
Mechanical stress induced by the nonselective beta-adrenergic agonist isoproterenol (5?mg/kg body weight) given daily for 10?days resulted in reduced fractional shortening and increased cardiac fibrosis in SJL/J mice as compared to controls.
      
Connective tissue growth factor: a crucial cytokine-mediating cardiac fibrosis in ongoing enterovirus myocarditis
      
 

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