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bladder carcinoma cell
Expression of a mutant hTERT in human bladder carcinoma cell line T24 and its clinical significance
      
After transfecting the fusion gene into bladder carcinoma cell line T24 by calcium phosphate-DNA coprecipitation, the steady expression of GFP-hTERT fusion protein was tested by fluorescent light microscopy.
      
The proliferation changes of bladder carcinoma cell line T24 were detected by light microscopy and senescence correlated β-galactosidase staining.
      
Conclusion: The mutant-type hTERT gene suppresses the proliferation of bladder carcinoma cell line T24 by competitive effect on telomerase activity.
      
Apoptosis induced by ginsenoside Rg3 in a human bladder carcinoma cell line
      
Erythroid progenitor cells (BFUe: burst forming units-erythroid) are cultured in 1 ml of 0.35% agarose in IMDM with 30% FBS and conditioned medium from human bladder carcinoma cell line 5637 or recombinant interleukin 3.
      
Three-dimensional modeling of T-24 human bladder carcinoma cell line: A new simulated microgravity culture vessel
      
Antiproliferative effect of hu-interferon-gamma in 674V and J82 bladder carcinoma cell lines
      
Stability of lectin binding properties expressed by human bladder carcinoma cell lines passaged in vitro or in nude mice
      
aFGF can also induce the motility of a rat-derived bladder carcinoma cell line (NBTII).
      
SEM and TEM studies revealed adherence and phagocytosis of BCG by the T24 human bladder carcinoma cell line in vitro.
      
Cytokine production by the human bladder carcinoma cell line T24 in the presence of bacillus Calmette-Guerin (BCG)
      
Cytokine secretion of a human bladder carcinoma cell line T24 treated with BCG was investigated.
      
Establishment and characterization of a multidrug-resistant human bladder carcinoma cell line RT112/D21
      
A doxorubicin-resistant human bladder carcinoma cell line RT112/D21 was established by continuous exposure of the parental line RT112 to increasing concentrations of doxorubicin over a period of 9 months.
      
Therefore, the mechanism underlying c-myc overexpression was further investigated in six bladder carcinoma cell lines.
      
The mRNA of Cox-1 and Cox-2 was expressed in both cultured bladder carcinoma cell lines.
      
Natural antibody to a human bladder carcinoma cell line
      
Sera from 144 healthy blood donors were screened for the presence of antibody against the bladder carcinoma cell line RT4 by means of an immune adherence assay.
      
A total of 143 patients with transitional cell carcinoma of the urinary bladder were tested for lymphocyte-mediated cytotoxicity against the bladder carcinoma cell line T24.
      
 

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