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cold injury
Myofibroblasts in subepicardium after local cold injury
      
Bioluminescence imaging of ATP, glucose, and lactate was performed in serial tissue sections at 4 h (n=4), 12 h (n=4) and 24 h (n=4) after cold injury or sham surgery.
      
Experiments also demonstrated that mortality of the larvae maintained in an extended supercooled state at -23°C was due to cold injury rather than freezing.
      
The metacarpal phalangeal joints and wrists were spared in all patients, and fingers which had been protected from the initial cold injury were similarly not affected.
      
A brief (2-h) exposure to 0°C elicits a protective response against subsequent cold injury at-10°C in the temperate flies and in B.
      
The capacity to undergo rapid the cold-hardening response (RCH) has been documented in diverse groups of insects and functions to protect against non-freezing cold injury and to preserve physiological performance in response to environmental cooling.
      
Cellular accumulation of sulphatides after intracerebral and endoneurial injections of the lipid was shown to occur if mesenchymal cell proliferation is induced by cold injury to the brain or by transection of peripheral nerves.
      
Brain edema was produced in dogs with cold injury, as described byKlatzoet al.
      
In the edematous white matter near the cold injury in cerebral cortex, serum proteins were distributed diffusely so that no relationship to the histological structures was noted.
      
Protein and enzyme changes in the edematous white matter and the CSF were studied during the course of experimental cold injury edema.
      
Reestablishment of the blood brain barrier to peroxidase following cold injury to mouse cortex
      
The catecholaminergic nerve supply to small intracerebral vessels following a cold injury to the mouse cortex
      
Vasogenic brain edema was induced in cats by cold injury (six animals), brain tumors (five animals), and brain abscesses (six animals).
      
Neuronal integrity and astrocytic reaction in cold injury: an immunohistochemical investigation
      
As a positive control two rats were subjected to cold injury.
      
Compared to other pathological conditions with increased blood-brain barrier permeability such as cold injury, the here observed serum albumin extravasations are very modest and, moreover, reversible.
      
Brain tissue from normal guinea pigs and from a cat subjected to cortical cold injury were used in this study.
      
The obvious hazard of a cold exposure under natural as well as artificial conditions is tissue cooling and the associated sequel of more or less harmful effects from cold injury to discomfort.
      
This study compares male and female contact cooling responses in order to ascertain whether a particular sex is at a greater risk to cold injury.
      
Cold-induced vasodilatation (CIVD), a paradoxical cyclical increase in finger temperature upon cold exposure, is assumed to serve against cold injury.
      
 

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