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prostate
A comparative analysis of the proteins in prostate tissues of the patients operated for hyperplasia (n = 7) or cancer (n = 5) was performed aiming to search for protein diagnostic markers.
      
The possibility of using AGR2 as a diagnostic marker of prostate cancer is discussed.
      
Hypermethylation of RASSF1A was frequently found in most major types of human tumors including lung, breast, prostate, pancreas, kidney, liver, cervical, thyroid and many other cancers.
      
Analysis of a Regulatory Protein Isolated from the Bovine Prostate
      
Discussed from this point of view are alpha-fetoprotein, the carcinoembryonic antigen, and the prostate-specific antigen, which are used as tumor markers.
      
The cDNA Microarray Profiling of Protein Kinases and Phosphatases: Molecular Portrait of Human Prostate Carcinomas
      
Signaling Pathways That Control the Growth and Survival of Prostate Carcinoma Cells in the Absence of Androgens
      
Androgen-dependent human prostate adenocarcinoma cell line LNCaP was used to study the effect of androgen deprivation on the cell response to TNF-related cytokines.
      
The results suggested a role of cytokines in the survival of prostate adenocarcinoma cells deprived of androgens in vitro.
      
Calcitriol induces transcription of the placental transforming growth factor β gene in prostate cancer cells via an androgen-ind
      
Calcitriol (1α,25-dihydroxycholecalciferol) suppresses the growth of prostate cancer cells.
      
Growth suppression of hormone-sensitive LNCaP prostate cancer cells by calcitriol is believed to depend on androgens, but the mechanisms of the interactions between the calcitriol-and androgen-dependent signaling pathways is unclear.
      
It was assumed that calcitriol stimulates production of PTGF-β independently of 5α-dihydrotestosterone and that its effect on prostate cancer cell growth is partly mediated by an androgen-independent mechanism.
      
Aberrant methylation of p16, HIC1, N33, and GSTP1 in tumor epithelium and tumor-associated cells in prostate cancer
      
The methylation status of p16, HIC1, N33, and GSTP1, which are involved in prostate carcinogenesis, was studied in prostate tissue samples containing neoplasms.
      
The methylation status of p16, HIC1, N33, and GSTP1 was also assessed in prostate biopsy material and operative tumor samples without laser capture microdissection.
      
Development of a biochip with an internal calibration curve for quantitating two forms of the prostate-specific antigen
      
Three-dimensional gel-based biological microchips were developed for simultaneous quantitation of total (PSAtot) and free (PSAfree) forms of the prostate-specific antigen in human serum in the "one patient, one biochip" format.
      
Loss of heterozygosity and microsatellite instability in tumor-associated stromal cells and tumor epithelium of prostate cancer
      
The epithelial and stromal components of prostate cancer (PC) and prostatic intraepithelial neoplasia (PIN) were isolated by laser capture microdissection and subjected to microsatellite analysis of chromosome regions 8p22, 13q14, and 16q23.
      
 

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