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chromatid recombination
Effects of the rad52 gene on sister chromatid recombination in Saccharomyces cerevisiae
      
Spontaneous and UV induced unequal mitotic sister chromatid recombination was examined in RAD+ and rad52-1 strains carrying the LEU2 gene inserted in the rDNA locus.
      
The rad52-1 mutation does not affect spontaneous sister chromatid recombination but greatly reduces the frequency of UV induced sister chromatid recombination.
      
Inhibition of thymidylate biosynthesis induces mitotic unequal sister chromatid recombination in Saccharomyces cerevisiae
      
Hybridization patterns obtained by Southern blot analysis of DNA from the leu+ and leu- sectors were consistent with the occurrence of unequal sister chromatid recombination.
      
Both rad6 mutants are proficient in spontaneous and UV induced unequal sister chromatid recombination in the reiterated ribosomal DNA sequence and are deficient in UV induced mutagenesis.
      
We describe here the chromosomal distribution of sister chromatid exchanges (SCEs) in four human tumor cell lines (two melanomas and two astrocytomas), and have mapped the sister chromatid recombination (SCR) sites.
      
Third, the DSB repair observed in mek1 dmc1 diploids is dependent on RAD54, a gene required primarily for sister chromatid recombination in meiosis.
      
 

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