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ischemic brain injury
Thus, choline succinate demonstrated a neuroprotective effect in conditions of ischemic brain injury.
      
The failed neuroprotective trials of the past have greatly increased understanding of the fundamental biology of ischemic brain injury and have laid a strong foundation for future advance.
      
Moreover, the recent favorable results of human clinical trials of hypothermia in human cardiac arrest and global brain ischemia have validated the general concept of neuroprotection for ischemic brain injury.
      
Animal stroke models have been extensively applied to advance our understanding of the mechanisms of ischemic brain injury and to develop novel therapeutic strategies for reducing brain damage after a stroke.
      
Many advances have occurred regarding an increased understanding of the basic pathophysiology of ischemic brain injury that could lead to enhanced therapy for this disorder.
      
This article summarizes recent insights into perinatal hypoxic-ischemic brain injury in the neonate.
      
The failed neuroprotective trials of the past have greatly increased understanding of the fundamental biology of ischemic brain injury and have laid a strong foundation for future advance.
      
Moreover, the recent favorable results of human clinical trials of hypothermia in human cardiac arrest and global brain ischemia have validated the general concept of neuroprotection for ischemic brain injury.
      
The clinical syndrome of TIA that is associated with ischemic brain injury is called "transient symptoms with infarction" (TSI).
      
The results indicated that TRPM7 channels in the ischemic cortex and hippocampus in rats might play a key role in ischemic brain injury.
      
The protective effect of rosuvastatin on ischemic brain injury and its mechanism
      
To study the protective effect of rosuvastatin on ischemic brain injury and its mechanism, focal cerebral ischemia/reperfusion was induced by occlusion of the middle cerebral artery (MCA) using the intra-luminal filament technique.
      
Consequently, the mechanisms of rosuvastatin's neural protection on ischemic brain injury are to enhance expression of eNOS, to inhibit expression of iNOS and activated caspase-3.
      
Here the evidence of the involvement of chemokines in ischemic brain injury is reviewed.
      
Although comparable detailed analyses of lymphocyte involvement in ischemic brain injury have not been performed, emerging findings suggest a role for T-cells in the pathogenesis of ischemic stroke.
      
Soluble Neuroprotective Antioxidant Uric Acid Analogs Ameliorate Ischemic Brain Injury in Mice
      
Estradiol exerts neuroprotective actions against ischemic brain injury
      
Neuroprotective effect of an antioxidant in ischemic brain injury
      
Because rods form rapidly in response to anoxia, they could also contribute to synaptic deficits associated with ischemic brain injury (e.g., stroke).
      
Multiple actions of the coumarine derivative cloricromene and its protective effects on ischemic brain injury
      
 

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