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hepatitis patients
Emergency adult living-donor liver transplantation is an effective treatment for fulminant hepatitis patients and is relatively safe for donors.
      
Distribution of ad and ay subtypes of hepatitis B surface antigen among hepatitis patients and symptomless carriers in Hungary
      
A total of 176 HBsAg positive sera from acute and chronic hepatitis patients and symptomless carriers (blood donors) in Hungary were subtyped for ad and ay determinants.
      
Two hundred and twenty sera from asymptomatic carriers and 129 sera from acute hepatitis patients were subtyped.
      
The incidence of subtypeayw in acute hepatitis patients was seen to be increasing.
      
These data suggest that the prognostic value of HBeAg in acute hepatitis patients may be taken into account when HBeAg persists in the serum and that anti-HBe does not invariably protect from the development of chronic hepatitis.
      
During one calendar year (September 1978 to August 1979) 14.3% of sera from hepatitis patients was positive for anti-HAV IgM.
      
Serological and tissue markers of HBV infection in chronic active hepatitis patients and in healthy carriers of HBsAg
      
drug addiction was found to have changed the epidemiological situation by decreasing the mean age of hepatitis patients, by increasing the male preponderance of HB and NANB patients, and by increasing the relative number of NANB cases.
      
8%) and HCV infection incidence (43.1%) than hepatitis patients (4.3% and 5.3%), IHS infants (6.0% and 8.0%) and daycared children (2.9% and 2.9%).
      
In the present study, the specificity of glycocholic acid was high in the detection of chronic active hepatitis patients at different cut-off levels.
      
From 200 acute viral hepatitis patients serum samples were collected successively, at various intervals.
      
Influenza vaccination in HBsAg positive chronic active hepatitis patients treated with interferon
      
The highest rates of mutation were found in chronic hepatitis patients and their family members.
      
However, the quality of life and the survival rate improve after transplantation in chronic viral hepatitis patients.
      
Among cirrhotic patients OC caused a 132% increase in FHNC (p >amp;lt; 0.05) and among the hepatitis patients a 69% increase (p >amp;lt; 0.05).
      
OC increased fasting glucose and insulin in the hepatitis patients (p >amp;lt; 0.01 and p >amp;lt; 0.001, respectively) and in the cirrhosis group (p >amp;lt; 0.01 and p >amp;lt; 0.05, respectively).
      
Alanine stimulation increased glucose in hepatitis patients after OC (p >amp;lt; 0.05) and after LC (p >amp;lt; 0.01).
      
During stimulation cortisol was higher following LC in hepatitis patients (p >amp;lt; 0.01) and cirrhotic patients (p >amp;lt; 0.05).
      
Fasting PGE2 was down-regulated after LC in hepatitis patients (p >amp;lt; 0.05) and cirrhotic patients (p >amp;lt; 0.01) and after OC in the hepatitis group (p >amp;lt; 0.001).
      
 

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