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clinical examination
A comprehensive posturographic (PG), electroencephalographic, and clinical examination was performed during rehabilitation in 17 patients (mean age 27.5 ± 7.4 years) who had suffered a severe craniocerebral injury (SCCI).
      
The results of comprehensive clinical examination and molecular cytogenetic analysis of a patient carrying chromosome 3p+ in 69% of the peripheral blood lymphocytes are presented.
      
Upon clinical examination, we found blue, shiny, bulging and imperforated hymen.
      
The patient had abdominal enlargement and clinical examination showed the hepatomegaly +5 cm.
      
All subjects who seemed suspicious of neuropsychiatric pathology were then given an individual clinical examination.
      
To establish the diagnosis of brain death as a valid and reliable diagnostic procedure, the clinical examination has to follow a standardized protocol.
      
Patient's history and clinical examination are the most important measures to identify at-risk patients.
      
Already clinical examination showed a whirring palpable over the chest associated with a diastolic accented continuous murmur and large, bounding arterial pulses.
      
By taking the patients' history and performing a clinical examination, first hints on the bleeding source can be gained.
      
The aftermath is neglect of careful clinical examination.
      
Diagnosis of Marfan syndrome is currently based on detailed clinical examination and/or mutation analysis in the fibrillin gene.
      
The clinical examination was normal except for a murmur at the apex of the heart.
      
The clinical examination revealed a lymph-node sarcoidosis of the chest.
      
The clinical examination revealed the palpable tumor showing characteristic X-ray findings.
      
Clinical examination revealed distal sensory inpairment, complaints of burning and lancinating extremity pains, ataxia and a decrease of deep tendon reflexes with total ankle jerk loss.
      
Clinical examination revealed distal sensory inpairment, complaints of burning and lancinating extremity pains, ataxia and a decrease of deep tendon reflexes with total ankle jerk loss.
      
The effects were evaluated from electromyographic (EMG) and kinematic analysis of treadmill walking with a portion of body weight supported, and from clinical examination and subjective assessments.
      
Both measurement and clinical examination showed the frequent occurrence of left-right asymmetry of reflex amplitudes.
      
The reliability of clinical examination of the tendon reflexes was examined by studying inter-observer agreement.
      
Clinical examination revealed sensorimotor polyneuropathy, predominantly affecting the lower extremities, hepatomegaly, and skin haemangiomas.
      
 

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