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apoptosis rate
Only NPSLE patients exhibited an increased neglect-apoptosis rate after incubation with culture medium; however, the neglect-apoptosis rate was associated with lymphopenia in both series of patients.
      
After lymphocyte incubation with autologous serum, only NPSLE patients exhibited a significant negative correlation between the neglect-apoptosis rate and the number of peripheral lymphocytes.
      
The total apoptosis rate is positively proportional to reaction duration and concentration of quercetin used.
      
The maximum apoptosis rate being (88.76±2.35)% was obtained when the concentration was 50.0 μmol/L and the cells were treated with quercetin for 72 hours.
      
The highest apoptosis rate was found to reach 13.38% by flow cytometry at 6 h after EMP irradiation.
      
Furthermore, apoptosis rate and cell cycle profiles with FCM had no significant change between MCA and monolayer cells.
      
Taxol exposure caused significantly decreased apoptosis rate in MCA cells than monolayer cells (P=0.012).
      
EPI-CDMN associated with PEMFs could significantly inhibit the proliferation of BIU-87 cells and induce cells apoptosis, the growth inhibitory rate and apoptosis rate were (21.82±3.18)% and (16.79±3.37)%, respectively.
      
The cell cycle and apoptosis rate were analyzed by FCM.
      
The apoptosis rate of bone marrow CD34+ cells induced by HHT was assayed with flow cytometer.
      
The apoptosis rate was detected by flow cytometry (FCM), the protein expression levels of bcl-2 and p53 in Raji cells were examined by SP immunohistochemistry.
      
The SASMC apoptosis rate in all groups was declined after treatment with SMI, but the effect of aminophylline was not obvious.
      
Before and after HepG2 and SMMC-7721 were treated with sTRAIL protein with various concentrations, the apoptosis rate was observed by using flow cytometry andin situ terminal deoxynucleotidyl tranferase (TdT) labeling.
      
After treatment with TRAIL (100 ng/ml) for 24 h, the apoptosis rate of HCC cells, Jurkat cells and human cholangiocarcinoma cell line QBC939 was 10%, 70%, 50% respectively.
      
The results showed that in cells transfected with pwtp53 and pwtp53+pSUMO-1, the apoptosis rate was (16.79±1.62)% and (18.15±1.36)% respectively, while transfected with pwtp53+pMDM2, the rate was decreased to (5.17±1.23)%.
      
The apoptosis rate was (14.06±1.84)% in the cells transfected with pwtp53+pMDM2+pSUMO-1, significantly higher than that in the cells Transfected with pwtp53+pMDM2 (P>amp;lt;0.01).
      
The results of flow cytometry and MTT colorimetry demonstrated a time- and concentration-dependent relationship between cell apoptosis rate and TRAIL concentration.
      
The apoptosis rate of U251 cells induced by p53 gene transfection was 17.38%.
      
The apoptosis rate was also significantly increased (17.80%, 20.03%, 22.34%) after combined treatment of p53 and irradiation with different doses (3, 6, 9 Gy).
      
Twenty-four h after irradiation, the changes in the chk1 protein expression was assayed by Western blot, and the cell cycles and apoptosis rate detected by FCM.
      
 

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